Supplemental Figure 3 – Pollen tube arrival and burst at the receptive synergid cell.

Jul 21, 2014

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Supplemental Figure 3 – Pollen tube arrival and burst at the receptive synergid cell.

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Supplemental Figure 3 – Pollen tube arrival and burst at the receptive synergidcell. Emasculated 450.3::GFP flowers were pollinated with LAT52DsRed pollen as in Figure 4. White arrows point to the last point where the pollen tube was clearly visible. Pollen tube rupture has occurred at least at this point or beyond. Scale bar =10µm.

Governing Equations

-1. Full Cell VOF 1. Partial Cell VOF . Empty Cell VOF 0. 2. 1. 0. Penn State Computation Day. Staggered Mesh. Computational Setup. Tube Wall ( r = R ). radial velocity, u. (i, j+1). (i+1, j+1). Computational grid for axisymmetric motion of a drop in a cylindrical tube.

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Supplemental Figure 1 .

Supplemental Figure 1 . mGlu1 α Receptor. A. IgG. 100. 80. DAPI. Co-localization of markers/CTX (%). 60. B. GM1. Transferrin receptor. 40. 20. Transferrin receptor. GM1. CTX. Supplemental Figure 2. A. Tat-blocking. Tat-mutant. Control. IP : mGlu1 α. Caveolin. mGlu1 α.

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Supplemental Figure 1

Supplemental Figure 1. BMP2 expression at 24 hours post transduction in ovine MSCs.

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Supplemental Figure 1

Supplemental Figure 1. Nomoxia. CH. Supplemental Fig 1. Vascular remodeling in rats for pulmonary arterial hypertension (PAH ). Weigert’s elastic staining revealed medial thickening changes in lungs . CH, chronically hypoxic . Supplemental Figure 2. ET A R. b -actin. CH. Normoxia.

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-- 1353/1078/872 -- 603 -- 310 -- 281/271 -- 234 -- 194 -- 118 -- 72

Supplemental Figure 1 A and B: Specificity validation of the PCR products Supplemental Figure 1A: Agarose gel electrophoresis of qPCR amplicons Gel strength: 4% NuSieve GTG (FMC Bioproducts, Rockland, ME) low melting temperature agarose

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Supplemental Figure 1

Supplemental Figure 1. A. Mock. MOI-100. MOI-10. 0.5%. 79.7%. 21.8%. CD3 PerCP. NY-ESO-1 Tetramer PE. B. C. Supplemental Figure 2. A. Thymus. L Humerus. R Humerus. Muscle. L Femur. R Femur. C. B. P < 0.0001. Supplemental Figure 3. A. B.

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Supplemental figure 1

Supplemental figure 1. 60. 40. 20. 0. control. MBZ 0.2 μ M. MBZ 0.1  M. A. B. Con MBZ 0.1 0.2 m M 72h. P S P S P S. a - a Tub. % polymerised tubulin. a - b Act. Supplemental figure 2. previous exp.

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Receptive Field

Receptive Field. [Green]. Receptive Field. The two-point threshold for any part of the body is determined by the size of the receptive fields and the extent of overlap.

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Supplemental Figure 2

Supplemental Figure 2. Supplemental Figure 2. Combined use of crizotinib or TAS-115 with erlotinib reverses resistance to EGFR-TKI induced by exogenous HGF on HCC827 cell.

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Supplemental Figure 2

Supplemental Figure 2. FEMALES. MALES. A. **. **. **. **. BARNES. 3. 3. *. ##. M. M. B. $. $. 18. M. C. *. HPWM.

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Supplemental Figure 1.

Supplemental Figure 1. A. GCN1L1. B. DSP. C. RCN3. D. AKR1B10. E. PDIA4. F. CTSB. Supplemental Figure 1. Cont. G. EIF5A. H. SERPINB5. I. TPD52. Supplemental Figure 2. A. Biological process. B. Cellular component. Supplemental Figure 3. A. C. B. A549. Cont. Sh1. Cont. Sh1.

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Supplemental Figure 3

Supplemental Figure 3: Relationship between transcript heritability H and eQTL sum R 2 plotted on hexbin graphs.

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Figure 9.1 U-tube manometer.

Figure 9.1 U-tube manometer. Figure 9.2 Well-type manometer. Figure 9.3 Inclined manometer. Figure 9.4 Mercury barometer. Figure 9.5 Bourdon gage. Figure 9.6 Dead-weight tester. Figure 9.7 Strain-gage pressure transducer. Figure 9.8 LVDT pressure transducer.

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Tsutsumi et al., Supplemental Figure 1

80. 40. 80. 60. 0. 40. 10 0. 10 1. 10 2. 10 3. 20. Negative control. GA-FITC. GA-FITC + DMAG-N-oxide. 0. 10 0. 10 1. 10 2. 10 3. Negative control. mock. DMAG-N-oxide. Tsutsumi et al., Supplemental Figure 1. a. Cell Number (%). Fluorescence Intensity (log). b.

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Kraenzlin et al. Supplemental Figure 1

µV. 2.0e+5. PYD. 1.5e+5. ISTD. Fluorescence (295/400 nm). 1.0e+5. DPD. 0.5e+4. 0.0. -0.5e+4. 2. 4. 6. 8. 10. 12. Time (min). Kraenzlin et al. Supplemental Figure 1. Supplemental Figure 1:

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Figure S1 Rink et al 2009

A. B. C. D. 30. LMCA. RMCA. a. 20. Percent hemispherical lesion volume. 10. E. 0. TTC. MRI. iNBO. iHBO. supplemental O 2 during ischemia. RA (control). rNBO. rHBO. supplemental O 2 at reperfusion. Figure S1 Rink et al 2009.

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Supplemental figure 1

2.5. *. 2.0. 1.5. LC3-II protein level. 1.0. 0.5. Ctrl. Pi. Supplemental figure 1. a. b. Ctrl Pi (3 mM). LC3I. LC3II. 50.  -actin. *. 40. 30. %Cells with GFP-LC3 puncta. 20. 10. Ctrl. Pi.

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Supplemental Figure 1 .

Supplemental Figure 1. mGlu1 α Receptor. A. IgG. 100. 80. DAPI. Co-localization of markers/CTX (%). 60. B. GM1. Transferrin receptor. 40. 20. Transferrin receptor. GM1. CTX. Supplemental Figure 2. A. Tat-blocking. Tat-mutant. Control. IP : mGlu1 α. Caveolin. mGlu1 α.

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Supplemental Figure 1

2500. 2000. 1500. 1000. 500. Supplemental Figure 1. CS-0777 product [ ng/mL ･ mg protein ]. 0. #1. #2. #3. #4. #5. Human specimen number. Supplemental figure 1. Examination of individual specificity of CS-0777-P phosphatase activity

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Supplemental Figure 1

Supplemental Figure 1. A. B. Supplemental Figure 1. (A) Dose response curves from MTT assays at 6 days of treatment with indicated concentrations of ICG-001. (B) IC50 values for PDAC cell lines after treatment with ICG-001.

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