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VARIABLE GENE EXPRESSION IN DIFFERENT HUMAN ORAL CANCER CELLS

The following slides contain guidelines using examples taken from the components of a sample abstract. Adhering to these guidelines will expedite the production of the list of abstracts within the limited time we have. VARIABLE GENE EXPRESSION IN DIFFERENT HUMAN ORAL CANCER CELLS.

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VARIABLE GENE EXPRESSION IN DIFFERENT HUMAN ORAL CANCER CELLS

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  1. The following slides contain guidelines using examples taken from the components of a sample abstract. Adhering to these guidelines will expedite the production of the list of abstracts within the limited time we have.

  2. VARIABLE GENE EXPRESSION IN DIFFERENT HUMAN ORAL CANCER CELLS Allison Yen1*, Nathan Overlid2 1Doctor of Dental Surgery Program, 2Department of Microbiology, University of the Pacific, Arthur A. Dugoni School of Dentistry, San Francisco As shown above, the abstract title should be in Times New Roman 12 point bold font and CAPITALIZED.

  3. VARIABLE GENE EXPRESSION IN DIFFERENT HUMAN ORAL CANCER CELLS Allison Yen1*, Nathan Overlid2 1Doctor of Dental Surgery Program, 2Department of Microbiology, University of the Pacific, Arthur A. Dugoni School of Dentistry, San Francisco The first and last names of the authors are in Times New Roman 12 point font with the affiliation numbers in superscript. The presenting author is indicated with an asterisk. There should be no degrees after the author names.

  4. VARIABLE GENE EXPRESSION IN DIFFERENT HUMAN ORAL CANCER CELLS Allison Yen1*, Nathan Overlid2 1Doctor of Dental Surgery Program, 2Department of Microbiology, University of the Pacific, Arthur A. Dugoni School of Dentistry, San Francisco The affiliations are in italics with the corresponding numbers in superscript before the affiliation. Please note the format of the student affiliations.

  5. 1Doctor of Dental Surgery Program, 2Department of Microbiology, University of the Pacific, Arthur A. Dugoni School of Dentistry, San Francisco Please note the section headings in bold, followed with a colon. OBJECTIVES: Survivin, a novel member of the inhibitor of apoptosis (IAP) protein family, is associated with malignant transformation and is over-expressed in oral squamous cell carcinoma (OSCC), but is undetectable in most normal adult tissues. METHODS: KB cells derived from epidermoid carcinoma of the nasopharynx, HSC-3, H357, and H413 cells derived from SCCs of the tongue, and non-tumor...... RESULTS: In all five cell lines, significantly higher level of luciferase activity was driven by the CMV promoter when compared.... CONCLUSIONS: The inability of the survivin promoter to drive high-level gene expression appears to preclude its potential use in gene therapy. The rationale for the different susceptibility to transfection observed with different cells may in....

  6. Please acknowledge any financial support for the work, and indicate if it hasbeen presented at a scientific meeting. CONCLUSIONS: The inability of the survivin promoter to drive high-level gene expression appears to preclude its potential use in gene therapy. The rationale for the different susceptibility to transfection observed with different cells may in.... This work was supported by Research Pilot Project Award 03-Activity 040 from the Arthur A. Dugoni School of Dentistry, and an AADR Research Fellowship (A. Yen). This work was presented at the 85th General Session of the International Association for Dental Research and 36rd Annual Meeting of the American Association for Dental Research, March 21-24, 2007, New Orleans, LA.

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