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Presented by: Toni Downen RN, MSN, CEN. Shock. Objectives. Define shock Discuss the nursing assessment of the patient in shock Identify classifications of shock Identify early and late signs of shock Treatment priorities in shock. Definition of Shock. Inadequate tissue perfusion and
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Objectives • Define shock • Discuss the nursing assessment of the patient in shock • Identify classifications of shock • Identify early and late signs of shock • Treatment priorities in shock
Definition of Shock Inadequate tissue perfusion and Cellular hypofunction/ hypoxia
Primary Goal • Maintain perfusion to the heart, brain, and lungs • These organs are perfused until the blood pressure decreases below 50
Classification of Shock • Hypovolemic shock • Low intravascular volume (Most Common) • Distributive shock • Maldistribution of blood volume • Cardiogenic shock • Poor pumping ability of heart • Dissociative or Obstructive shock • Inability of blood to deliver oxygen
Stages of Shock • Compensated Shock • Sympathetic Nervous System response to decreased tissue perfusion • Uncompensated Shock • Compensatory mechanisms no longer able to to maintain tissue perfusion • Irreversible (Refractory) Shock • Progression to cellular, tissue, and organ death
Early Signs of Shock • Orthostatic vital signs • An orthostatic systolic decrease of 10 to 20 millimeters of mercury or increase in pulse of 15 beats/min is considered "significant." • Take orthostatic vital signs recumbent and after standing for 1 to 2 minutes. • Decreased Urinary Output-less than 30 Ml/hour • Respiratory alkalosis • Restlessness, anxiety
Early Signs of Shock • Tachycardia • Attempts to compensate for decreased cardiac output • Altered mental status • The brain is the most sensitive organ to alterations in cardiac output • Unable to concentrate • Difficulty following commands
LATESigns of Shock • Decreased systolic Blood Pressure • Metabolic acidosis • Decreased Level Of Consciousness • Cold clammy skin or mottling • Decreased Urinary Output (oliguria to anuria) • Decreased Cardiac Output
Hypovolemic shock • Inadequate tissue perfusion resulting from inadequate circulating volume • Blood loss • Burns
Treatment of Hypovolemic Shock • Slow or stop fluid or blood loss • For dehydration give fluids • 0.9 Normal Saline is the fluid of choice • Lactated Ringers may also be used • For blood loss • Give fluids until blood arrives
Distributive shock • Based on loss of vascular tone resulting in vasodilation and abnormal distribution of blood • Neurogenic Shock/ Spinal cord injury • Septic shock-endotoxins and inflammatory mediators causing vasodilation and other perfusion abnormalities
Septic Shock-2 Phases • Warm shock-(hyperdynamic) (1) • High cardiac output and low peripheral vascular resistance from vasodilation(bradykinins, histamine, etc.) • Capillaries become permeable causing leakage of fluid shifting (3rd spacing) • Fever and increased respiratory rate (due to infection causing bacteria) • Diuresis due to high osmotic load handled by the kidneys (due to dying bacteria) • Temporarily increases cardiac output and contractility –Warm phase can last 6-72 hours
Cold Phase (late stage) • Low output or high resistance shock • Late and nearly irreversible (can’t distinguish from hypovolemic shock) • Subnormal temperature and low white blood count • Profound hypotension and hypoperfusion • Skin cold and mottled everywhere • Pulse and respirations rapid, but cardiac output decreases • Leads to vasoconstriction of major organ circulation leading to organ death
Treatment of Septic Shock • Control infection source • Cultures and appropriate antimicrobials • Drain or debride infected tissue • Support Cardiovascular system and perfusion • FLUIDS • Vasoconstrictors to reverse massive peripheral vasodilation • Inotropic support to increase cardiac contactility (Dobutamine) • Control temperature
Treatment of Septic Shock • Improve oxygenation • Increase O2 delivery to compensate for high O2 consumption • Serum Lactate-To measure cellular perfusion: If lactate is greater than 4 mmol/L it is associated with mortality and shock • Prophylactic heparin, and blood components to treat coagulopathies
Neurogenic Shock • Cause: • Loss of sympathetic tone massive vasodilation • Spinal cord injury especially involving cervical or high thoracic vertebra • Spinal anesthesia • Vasomotor center depression from head trauma • Priorities • Maintain Airway • Stabilize spine • Support Respirations
Treatment of Neurogenic Shock • Correct hypovolemia with the minimum volume needed for systolic Blood Pressure greater than 90 • Vasopressor support for perfusion to constrict vasculature and increase blood pressure • Treat persistent bradycardia with atropine or pacing • Reverse hypoxemia • Maintain body temperature • Slowly rewarm • Remember Airway, breathing and circulation are always a priority and must be reassessed often
Cardiogenic shock • The heart fails to pump blood adequately to all parts of the body • Usually caused by: • Injury to the heart • Myocardial infarction • Electrical shock
Treatment of Cardiogenic Shock • Never forget OXYGEN! • Goal is to increase cardiac output • Correct preload • Increase contractility • Correct afterload
Decreasing Preload in Cardiogenic Shock • Nitroglycerin (Tridil) • commonly used, especially if there is concern that ischemia is an underlying or precipitating factor • Start at 10 to 20 mcg/min and increase by increments of 10 to 20 mcg/min every 5 minutes until the desired effect is achieved
Decreasing Preload in Cardiogenic Shock • Furosemide and other diuretics • Start with 20 mg Intravenous and observe response. • Titrate dose upward until adequate diuresis is established. • If the patient is receiving furosemide over a long-term give 1 to 2 times the usual daily dose by slow (over 1 to 2 minutes) IV bolus.
Increasing Contractility in Cardiogenic Shock • Dobutamine (2.5 to 15 mg/kg/min) • Dopamine (2 to 20 mg/kg/ min) • may be needed for pressure support or as a positive inotrope. • Consider pacing when bradycardia is thought to be the cause • Consider cardioversion for arrhythmias
Decreasing Afterload Cardiogenic Shock • Nipride or Beta blocker to decrease systemic vascular resistance • Consider an intra-aortic balloon pump if left ventricular failure is suspected • Surgery may be indicated under rare conditions such as valvular heart disease or rupture of ventricular septum
Dissociative or Obstructive Shock • Inability of Red Blood Cells to deliver oxygen due to obstruction of blood flow • Tension pneumothorax • Clot of a great vessel or Pulmonary Embolis • Cardiac Tamponade • Carbon monoxide poisoning • Abdominal Aortic Aneurism (AAA) • Gravid uterus
Treatment of a Tension Pneumothorax • Maintain Airway • Deliver High flow oxygen • Decompress effected side of the chest • Needle decompression • Chest tube placement • Support volume if blood or fluid is the cause
Treatment of a Pulmonary Embolus • Support Airway • High Fractional Inspired Oxygen (measured O2) • Cat Scan of the chest • Ventilation perfusion scan • Consider Tissue Plasminogen Activator • Treat with appropriate anticoagulants
Treatment of Cardiac Tamponade • Oxygen • IV & lab • Frequent vital signs • Antibiotics • Pericardiocentesis (Aspiration of fluid from pericardial sac) • Surgical pericardiectomy (removal of the sac)
Treatment In All Types of Shock • Remember Airway, Breathing and Circulation • These are your top priority • Establish adequate ventilation and oxygenation • Restore optimum circulation and volume • Identify type of shock and Treat underlying causes
Treatment In All Types of Shock • Maintain adequate cardiac output • Maintain urine output between 30 - 60 ml/hr. • Usually accomplished by fluids or inotropes • Maintain optimum internal metabolic environment (check your Arterial Blood Gases) • Keep patient warm
The End Questions?
References • McQuillan, Von Rueden, Hartsock, Flynn and Whalen. Trauma Nursing From Resuscitation through Rehabilitation (3rd edition) (2009) • Sheehy’s Manual of Emergency Care (7th edition) (2013) • Wahl, Sharon C. (2011) Septic Shock: How to Detect it Early