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Management of Bone Defects in TKA. Dr. Mohammad Hossein Dehghani Isfahan Jesus Hospital. Introduction. more common in revision TKA , BUT they do occur in primary TKA also. Causes of bone defects in primary TKA. erosion secondary to angular arthritic change,
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Management of Bone Defectsin TKA Dr. Mohammad HosseinDehghani Isfahan Jesus Hospital
Introduction • more common in revision TKA, BUT • they do occur in primary TKA also.
Causes of bone defects in primary TKA • erosion secondary to angular arthritic change, • inflammatory arthritis, • osteonecrosis, • and fracture.
Bone defects in primary TKA • typically :asymmetrical & peripheral, • although contained deficiencies caused by cyst formation may occur.
Bone defects in primary TKA • In primary TKA ,base of contained and peripheral defects : condensed sclerotic bone, • In revision surgery, removal of components often leaves osteopenic surfaces
Major Concern • Diminish of subchondral bone strength distal to the subchondralplate. • Solution:thelevel of lateral tibial resection should not exceed 1 centimeter to avoid compromising implant durability, • others have demonstrated that proximal tibial bone strength is adequate to 20 mm
Solution Right Wrong
Management • translation of the component away from a defect, • lower tibial resection, • cement filling, • autologousbone graft, • allograft, • wedges or augments, • custom implants..
Management(Use of stems ) • in primary TKA: • necessary when bone grafting is required • or when the bone defect compromises fixation and renders the resurfacing component unstable without the added support of intramedullary fixation
Management (Lateral Translation) • Lateralizing a smaller tibial component : effectively eliminates bony defect by removing contact of implant with defect • However, the largest tibial tray size and polyethylene insert should always be favored to create the largest reasonable contact surface to distribute load.
Management (Lower Tibial Resection) • is often effective • limit of a lower tibial resection is :insertion of the ITB and infrapatellarligament. (Gerdy) • Additionally, a lower tibial resection will complicate component fit because of the natural taper of the tibia, necessitating the use of a smaller tibial component or tapered tibial augments.
Management (Lower Tibial Resection) Right Wrong
Management (Cement Filling) • Lotke &Ritter demonstrated satisfactory long-term results with cement fill provided: • tibialbone defects are no deeper than 20 mm • and involve less than 50% of either plateau. But • cement fill with or without screw reinforcement is an inferior method of defect management & radiolucent lines are commonly observed under defects filled with cement. • larger volumes of cement introduce the risk of thermal necrosis of the cement-bone interface
Management (Cement Filling) Step-cut filled with cement (under tibialcomponent)
Management(autologous bone graft) • readily available in primary TKA. • high rates of incorporation • osteoinductive properties • lack of potential disease transmission • typically used when the size criteria for cement fill are exceeded.
Management (criteria that promote improved outcome) • creating viable/bleeding bed of host bone, • proper fit and finish of graft in host bed, • complete coverage of graft by the component to avoid graft resorption secondary to stress shielding, • optimal alignment of components for even load distribution, • limited weight bearing when larger grafts are used to allow for graft union, • and grafts protected with stems when required .
Management • Contained defects : easily filled with bone graft, • Peripheral defects : more challenging.
Management(Custom Prostheses and Metal Wedge Augmentation) • Metal wedge : intraoperative construction of a custom implant to address a bone defect, • Defects of less than 25 mm • Custom prostheses : for dealing with larger defects ( > 25mm) • limitations of practicality and cost
Metal Wedge Augmentation • available in triangular and rectangular shapes, • both cemented and cementless options. • load transfer across a larger defect: a rectangular block and stem augmentation. • good results using wedges attached with screw fixation
Distal Femoral Defects • frequently observed in valgus deformities when the lateral femoral condyle is dysplastic. • As with the tibia, defects can be managed with cement, bone graft, and metal augments.
Build-up required Condylar resection
Contained Defects: managed in the same manner as contained tibial defects. • Peripheral Defects : • affecting the chamfer cuts, • affecting the distal surface, or • causing major bone loss.
Femoral deficiencies • increasing stages of bone loss: • Stage 1 • when the femoral osteotomy includes a portion of the lateral distal femur, but contouring to accommodate the femoral component results in chamfer “air cuts” anteriorly and posteriorly. • cement fill is acceptable for filling anterior and posterior spaces between bone and prosthesis. The sclerotic bone surface should be prepared to accept cement interdigitation.
Femoral deficiencies • Stage 2 • occurs when the level of the femoral osteotomy passes distal to the lateral femoral condyle even without chamfer cuts. • In this situation, cement fill typically is unsatisfactory unless combined with a femoral stem extension. • Even in this instance, a metal augment to the distal femur is preferred.
Femoral deficiencies • Stage 3 • refers to massive bone loss of one femoral condyle. • Substantial bone loss can be managed with allograft or metal block augmentation, • Allograft requires a period of non–weight bearing postoperatively and a femoral stem extension. The advantage of allograft is that if a revision is required, bone stock may be partially restored. • Metal augments allow quicker rehabilitation without restricted weight bearing .
Femoral deficiencies • In general, optimized collateral ligament stability and restoration of normal anatomy is preferable to the use of constrained prostheses.