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Gain insights on the anatomy, physiology, aging-related changes, risk factors, diagnostic testing care, hemodynamic monitoring, and more. Discover the impact of aging, modifiable risk factors, clinical manifestations, and assessment techniques vital in cardiovascular nursing.
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Nursing Assessment of the Cardiovascular System Keith Rischer RN, MA, CEN
Today’s Objectives… • Review the anatomy and physiology of the cardiovascular system. • Describe cardiovascular changes associated with aging. • Identify factors that place patients at risk for cardiovascular problems. • Explain and describe pre- and post-care associated with diagnostic cardiovascular testing. • Explain the purpose of hemodynamic monitoring.
Right Coronary Left Anterior Descending Circumflex Coronary Arteries
Cardiac Conduction SA node Both Atria AV Node Both Ventricles Bundle of His Bundle Branches- Perkinje Fibers
Diastole Diastole Lower # in BP (120/80) Ventricles are relaxed Passively fill from atria
Systole Higher # in BP (120/80) Ventricles are contracting and emptying SBP accurately reflects afterload
Cardiac Output • CO = Stroke volume x heart rate • SV (80cc) x HR (80)= 6400cc (6.4 lpm) • Daily pumps 1800 gallons • 657,000 gallons every year • Over 80 year lifetime: • 52,560,000 gallons
Preload: Right side of heart Preload=primarily venous blood return to RA Right and left side of heart filling pressure (atria>ventricles) Pressure/Stretch in ventricles end diastole
Maximum efficency of CO achieved when myocardium stretched appx 2 ½ times length Think rubber band CO decreased with lower preload/filling pressures or too high Starling’s Law of the Heart
“AFTER” load: Left side of heart Force of resistance that the LV must generate to open aortic valve Correlates w/SBP
Contractility Ability of heart to change force of contraction without changing resting length Influenced by Ca++ Inotropic-Influencing contractility independent of Starling mechanism Positive inotropic Negative inotropic
Assessment Techniques • History • Demographic data • Age • Gender • Pre vs. post menopause • Family history and genetic risk • Personal medical history • DM, HTN • BCP use for women • Diet
Modifiable Risk Factors • Cigarette smoking • Physical inactivity • Obesity • Psychological factors • Chronic disease
Changes with Aging • Calcification in valves • Pacemaker cells decrease in number • Conduction time increases • Left ventricle increases • Aorta and large vessels thicken and become stiffer • Baroreceptors less sensitive
Women & CAD Vague-atypical chief c/o Only 53% have CP as chief c/o Fatigue chief c/o Typically develop CAD 10+ years later then men Mortality twice as high Less likely to have definitive 12 lead EKG Smaller coronary arteries Higher prevalence of silent ischemia CABG higher mortality rate/complications After first MI-more likely to suffer fatal event Women > 50 pay must address HTN, high cholesterol, family history, diabetes
Men Women Develop 10-15 years earlier Initial event AMI Classic CP sx Develop greater collateral circulation compared to women Influence of menopause…4x risk Causes more deaths in women than men Initial event angina Atypical CP sx…fatigue
Elderly & CAD More likely to have vague, atypical c/o SOB, fatigue, syncope or falls Any fall must be investigated for mitigating circumstances Less likely to have radiation
Clinical Manifestations:Dyspnea • Can occur as a result of both cardiac and pulmonary disease • Difficult or labored breathing experienced as uncomfortable breathing or shortness of breath • Dyspnea on exertion (DOE) • Orthopnea • dyspnea when lying flat • Paroxysmal nocturnal dyspnea (PND) • after lying down for several hours
Other Clinical Manifestations • Fatigue • Palpitations • Weight gain • Syncope • Extremity pain • Ischemia • Venous insufficiency
CV Physical Assessment • General appearance • Integumentary system • Skin color • Skin temperature • Extremities • Blood pressure • Orthostatic • Arterial pulses • Jugular venous distention
Precordium • Auscultation • Normal heart sounds…S1S2 • Paradoxical splitting • Gallops • S3 • S4 • Murmurs • Systolic most common • Pericardial friction rub
Serum Markers of Myocardial Damage • Troponin • B-Natruetic Peptide • Serum lipids • C-reactive protein • Lytes • K+ • Mg++ • Blood coagulation • PT & INR
Brain Natriuetic Peptide:BNP 95 % of BNP resides in ventricles As pressure inc in ventricles in HF-BNP is released Bodies own ACE/B-blocker Only lab test that measures HF Normal is less than 100 Elevated 100-500 + for CHF >500 Uses: dx- assess response to tx
Cardiac Catheterization • Client preparation • Possible complications: myocardial infarction, stroke, thromboembolism, arterial bleeding, lethal dysrhythmias, and death • Pre-procedure: • Review procedure (video) • Consent • NPO or light breakfast • Cath site shaved • Premeds - sedative
Cardiac Catheterization • Procedure • Pt awake – report any chest pain or symptoms • May proceed to Stent Placement • Follow-up care: • Restricted bedrest, insertion site extremity kept straight • Assess groin site and distal pulses closely • Monitor vital signs • Force fluids • Assess for complications
Other Diagnostic Tests • 12 lead EKG • Holter monitor (ambulatory) • Electrophysiologic (EP) study • Exercise Stress test • Echocardiography • Pharmacologic stress echocardiogram • Transesophageal echocardiogram • Thallium imaging