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How to give a talk. COMMUNICATION. What is the point of a “ talk ” ?. Specifically communicating scientific results, ideas
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COMMUNICATION What is the point of a “talk”? • Specifically communicating scientific results, ideas • Less about impressing them with the amount of work you’ve done, the complexity of mathematical models, the exoticness of your field site, the cuteness of your study organism….
Today’s talk What makes a good/bad talk? What makes you a good/bad talker? Mechanics & Style
Introduction Methods Results Discussion *but maybe don’t make a slide like this
Background Methods Results Future Directions
But first: acknowledgments! The People and the Money Nick Colegrave Sue Healy Tom Little Tom Tregenza
A good talk (1) The content!
A good talk The content! (a good story and some fun biology)
A good talk The content! (2) A confident speaker
A good talk The content! (2) A confident speaker (3) Clear, engaging presentation
A good talk The content! (2) A confident speaker (3) Clear, engaging presentation (4) Good timekeeping
Being a good speaker Be confident “Sharing” your work People want you to succeed
A bad talk A “bad” speaker (too fast, too quiet, too nervous, too cocky…)
A bad talk A “bad” speaker (2) Boring story (=no story?)
A bad talk A “bad” speaker (2) Boring story (=no story?) (3) Too long
A bad talk A “bad” speaker (2) Boring story (=no story?) (3) Too long (4) Unappealing slides
Being a good speaker Be confident: -look at the audience -speak to the audience -practice -go to other talks -never apologise…!
Telling a good story Have a narrative: Have a clear question and go somewhere with it -for example, pose a question, but then return to the question at the end, and make it clear if you have/have not answered it.
Mechanics & Style Actually giving a talk: (1) Timing (2) Speaking (3) “Stagecraft”
Timing Very important! 1 slide = 1 minute Use a watch Multiple places to STOP Be prepared to STOP
Timing Part of getting timing right is just being realistic about how much you can fit in 10-15 min talk = 1 experiment 20-30 min talk = 2-3 experiments 40-50 min talk = 2-3 sections* *give people a break…
Marks dip during sessions (2002) • This effect WORSE for tutors! (P<0.001)
Timing 10-15 min talk = 1 experiment 20-30 min talk = 2-3 experiments 40-50 min talk = 2-3 sections 60 min talk = what?!
Timing Introductions are nearly ALWAYS too long
Mechanics & Style: Speaking Important! LOUD and CLEAR Be natural Be prepared to alter how you speak
“Stagecraft” Important! Be yourself Try and be comfortable But… Perform, burn energy
Should I try and be funny? Crudgington and Siva-Jothy 2000
Should I try and be funny? Crudgington and Siva-Jothy 2000
Should I try and be funny? Crudgington and Siva-Jothy 2000
Should I try and be funny? ≠ RSPB images not RSPB images
Style and slides Keep it simple
Quiz time: TRUE or FALSE? PowerPoint is really great and never fails? FALSE
Powerpoint Make sure you test your talk on multiple computers, AND where you will give the talk Learn how to “embed fonts” “Insert” pictures c.f. drag & drop Compress picture sizes Email yourself your talk/put on website
Images: Decompress TIFF files of death… Cannot decompress TIFFTM file Cannot decompress TIFFTM file Cannot decompress TIFFTM file Cannot decompress TIFFTM file Cannot decompress TIFFTM file
Avoid tests of sex-linkage Anything wrong with this slide?
Easy things to forget Anything wrong with this slide?
Pictures versus text? Text can be an aide memoire Something to fall back on BUT Some people hate being told what is already on the screen…
Text: remember, you do want them paying attention to you Introduction Natural populations frequently show considerable genetic variation in immune functions (e.g. Kurtz & Sauer 1999; Ryder & Siva-Jothy 2001; Cotter et al. 2004) and pathogen resistance (e.g. Lively 1989; Henter & Via 1995; Carius et al. 2001; Koskela et al. 2002). This seems paradoxical because immune defence is closely associated with organisms’ fitness, and natural selection is expected to erode genetic variation in fitness related traits by eliminating the least fit alleles from populations (reviewed by Roff 1997). What then maintains genetic polymorphism in immune functions? One likely factor that can maintain such variation is the high diversity of pathogens the hosts are exposed to. When the outcome of infection depends on the combination of both host and pathogen genotypes (GH × GP interaction; see Schmid-Hempel & Ebert 2003) pathogens impose a heterogeneous selective pressure on their hosts, which promotes maintenance of genetic variation. GH × GP interactions have been described from several plant–pathogen (e.g. Burdon & Jarosz 1991; Salvaudon et al. 2005) as well as animal–pathogen (e.g. Dybdahl & Lively 1998; Carius et al. 2001) systems, suggesting that they play an important role in maintenance of genetic polymorphism in resistance. GH × GP interactions, however, can effectively maintain genetic variation only in defence mechanisms which are highly specific to different pathogen genotypes. Pathogen resistance is often not strictly specific, but consists of defences that are independent of the genetic identity of the pathogen (i.e. non-specific immune defence; Frank 2000, Moret 2003). This part of immune defence is typically controlled by several genes and evolves through selection on heritable additive genetic variance (Cotter et al. 2004; Schwarzenbach et al. 2005). Therefore, mechanisms other than GH × GP interactions are needed to explain the maintenance of high polymorphism in immune functions. It has been suggested recently that genetic variation in pathogen resistance could also be maintained by environmental heterogeneity and physiological trade-offs (reviewed by Lazzaro & Little 2009). Several facts