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بسم الله الرحمن الرحيم. THE FACIAL NERVE. Topics. Gross applied anatomy Pathology of nerve injury and regeneration Principles of the electrophysiological tests Causes of facial nerve paralysis Principles of treatment of facial paralysis Bell’s palsy. Facial Nerve Fibers. Motor
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Topics • Gross applied anatomy • Pathology of nerve injury and regeneration • Principles of the electrophysiological tests • Causes of facial nerve paralysis • Principles of treatment of facial paralysis • Bell’s palsy
Facial Nerve Fibers • Motor • to the stapedius and facial muscles • Secreto-motor (parasympathetic) • to the lacrimal gland and the submandibular and sublingual salivary glands • Taste • from the anterior two thirds of the tongue and palate • Sensory • from the external auditory meatus
Anatomical Divisions • Intracranial • Cranial (intratemporal) • Extracranial (extratemporal)
The Intracranial Part • Include the nuclei and the Cerebello Pontine angle semgents
Lower motor lesions affect all the ipsilateral facial muscles • Upper motor lesions spare the upper facial muscles and affect the contralateral lower face
Upper motor Lower motor
Facial Nerve Fibers • Motor • to stapedius, and facial muscles • Secreto-motor • to the submandibular, sublingual, and lacrimal glands • Taste • from the anterior two thirds of tongue and palate • Sensory • from the external auditory meatus
Clinical Manifestations • Paralysis of facial muscles • Asymmetry of the face • Inability to close the eye • Accumulation of food in the cheek
Clinical Manifestations • Paralysis of facial muscles • Asymmetry of the face • Inability to close the eye • Accumulation of food in the cheek • Phonophobia • Dryness of the eyes • Loss of taste
Neuropraxia (Conduction block) Neurotmeses (Degeneration)
Regeneration • In neuropraxia: restoration of full function if the cause is treated • In neurotmeses : No recovery unless the distal and the proximal parts approximated • Recovery is delayed and usually is incomplete
Principles of management of facial nerve paralysis • Care of the eye • Treatment of the cause if applicable • Treatment of the nerve varies according to the degree of the paralysis
Partial facial paralysis • Being partial means that some of the nerve fibres are in continuity and recovery is expected by conservative treatment (e.g. removal of pressure, steroid etc)
Complete facial paralysis • Complete paralysis may be a result of neuropraxia or/and degeneration • If it is due to neuropraxia, recovery is expected by conservative treatment • If it is due to degeneration, surgical treatment is required • To differentiate between degeneration and neuropraxia electrophysiological tests are required
Electrophysiological Tests • It detect degeneration of the nerve fibres • Useful only 48-72 hours following the onset of the paralysis
Electrophysiological Tests • Nerve Excitability Test (NET) • Electroneurography (ENoG)
Nerve excitability test (NET) • The current’s thresholds required to elicit just-visible muscle contraction on the normal side of the face are compared with those values required over corresponding sites on the side of the paralysis
Electroneurography (ENoG) • The amplitude of action potentials in the muscles induced by the maximum current is compared with the normal side ; and used to calculate the percentage of intact axons.
Interpretation of the tests • Not useful in the first 48 – 72 hours • After 48-72 hours (the time required for degeneration to take place) • Normal results means that there is no degeneration (Neuropraxia) • Abnormal results means degeneration
Topognostic Tests • Indicated in some cases to locate the site of the injury
Topognostic Tests • Schirmer's test • Test the lacrimation function
Topognostic tests • Schirmer's test • Stapedial reflex • Taste sensation
Topognostic tests • Schirmer's test • Stapedial reflex • Taste sensation
Causes of facial paralysis • Intracranial causes • Cranial (intratemporal) causes • Extracranial causes
Causes of facial paralysis • Congenital: Birth trauma • Traumatic: Head and neck injuries & surgery • Inflammatory: O.M, Necrotizing O.E., Herpes • Neoplastic: Meningioma, malignancy ear or parotid • Neurological: Guillain-Barre syndrome, multiple sclerosis • Idiopathic: Bell’s palsy
Congenital Facial Palsy • 80-90% are associated with birth trauma • 10 -20 % are associated with developmental lesions