1 / 18

Multiple Congenital Cardiac Anomalies Accession# 147266

Multiple Congenital Cardiac Anomalies Accession# 147266. Christina Copple , DVM Monday 2/28/2011. 10mth, Male, Pomeranian. Late January purchased from breeder with no known prior medical concerns Episode after a moment of activity --- fell on side, stiff, dilated pupils, unaware

leanna
Download Presentation

Multiple Congenital Cardiac Anomalies Accession# 147266

An Image/Link below is provided (as is) to download presentation Download Policy: Content on the Website is provided to you AS IS for your information and personal use and may not be sold / licensed / shared on other websites without getting consent from its author. Content is provided to you AS IS for your information and personal use only. Download presentation by click this link. While downloading, if for some reason you are not able to download a presentation, the publisher may have deleted the file from their server. During download, if you can't get a presentation, the file might be deleted by the publisher.

E N D

Presentation Transcript


  1. Multiple Congenital Cardiac AnomaliesAccession# 147266 Christina Copple, DVM Monday 2/28/2011

  2. 10mth, Male, Pomeranian • Late January purchased from breeder with no known prior medical concerns • Episode after a moment of activity --- fell on side, stiff, dilated pupils, unaware • Recovered within minutes • Pants when excited or playing • ER DVM: heart murmur & suspected PDA • Specialty clinic: findings more consistent with pulmonic stenosis • Referral to NCSU for further evaluation

  3. NCSU cardiology work-up • Grade III/VI left apical systolic murmur • Normal lung sounds • Echocardiogram • PCV/TS

  4. Echocardiogram- Rt parasternal short-axis view of ventricles at level of papillary muscles • Severe right ventricular hypertrophy • Flattening of interventricular septum

  5. Echocardiogram – M-mode through ventricles • Single narrow US beam of echoes as distance vs time • Provides time-dependent measurements • chamber dimension • RV hypertrophy • RV wall thickness should be 1/3-1/2 that of the LV • Lumen of LV normally ~3X diameter of RV lumen

  6. Echocardiogram – Rtparasternal long-axis 4 chamber view • RV hypertrophy, severe • RA enlargement, moderate

  7. Echocardiogram – Rt parasternal short-axis view at heart base of pulmonic valve (zoomed in) • Supravalvular pulmonic stenosis • Post stenotic dilation • Turbulent flow across stenosis

  8. Echocardiogram – Lt parasternal short-axis view of pulmonic valve (payme view)

  9. Echocardiogram – Lt parasternal short-axis view of pulmonic valve • Continuous wave Doppler signal • accurately evaluates high velocities without aliasing • Continuously sends and samples signal • spectral broadening expected as there is no discrimination between laminar vs turbulent flow

  10. Echocardiogram – Lt parasternal short-axis view of pulmonic valve • Maximum velocity • Utilize modified Bernoulli equation • 4V2 • determine presssure gradient • Presssure gradient ~ 130 mmHg = severe as it is > 80

  11. Echocardiogram – Lt parasternal apical 4 chamber view • RA enlargement, moderate • Mild tricuspid insufficiency

  12. Wait, There’s More!!

  13. Echocardiogram – Rtparasternal short-axis view of ventricles at level of papillary muscles • BONUS Lesion!! • VSD – apical position in muscular septum • With right-to-left shunting

  14. Echocardiogram – Lt parasternal apical 4 chamber view of VSD with color Doppler

  15. Contrast Echocardiogram – Bubble study with agitated saline!!

  16. Uncommon forms of pulmonic stenosis & VSD • Supraventricular pulmonic stenosis • Increased RVOT obstruction • Rare, less common than valvular – Giant Schnauzers • Apical VSD in muscular septum • Less common than perimembranous • Single opening in LV • Multiple openings in RV • Right-to-left shunt due to elevated right sided pressures • Decreased O2 content of systemic circulation • Humans – neonates and small infants: uncommon, usually present with heart failure & associated anomalies such as pulmonic stenosis, PDA, aortic coarctation, etc. • PCV = high normal • Compensatory • Episode either syncopal or cyanotic

  17. What now? • Balloon valvuloplasty? • Could help but….. • Might result in altered pressure differential between right and left sides • Result in Left-to-Right shunt  pulmonary overcirculation • Amplatzer of VSD? • Reduce potential for Left-to-Right shunt • Not commonly performed • Never performed at NCSU

  18. References • Fox, Philip R., Sisson, David, and Moise, N. Sydney. Textbook of Canine and Feline Cardiology Principles and Clinical Practice. 2nd ed. W.B. Saunders Company. Philadelphia, PA. 1999. • Kumar K, Lock JE, and Geva T. Apical Muscular Ventricular Septal Defects Between The Left Ventricle And The Right Ventricular Infundibulum. Diagnostic And Interventional Considerations. Circualtion. 1997. March 4; 95(5):1207-1213. • Ramesh, et al. Transcatheter Closure of Congential Muscular Ventricular Septal Defect. JIntervenCardiol. 2004; 17:109-115.

More Related