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Explore the anatomy and physiology of the nervous system, including neuron function, sensory and motor neurons, and the role of myelin. Understand neurologic disorders, diagnostic tests, and the nursing assessment of neurologic function. Learn about age-related changes in the nervous system and various types of neurologic diseases.
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NURS 1130 Adult Nursing III Unit I Chapters 27, 28, 29 Teresa Champion, RN, MSN 9/2012
Chapter 27 Neurologic Disorders
Neuron (Nerve Cell) Functional unit of the nervous system that conducts electrical impulses from one area of the brain to another Sensory neurons Transmit information from distal parts of the body or environment toward the central nervous system Motor neurons Carry motor information from the CNS to the periphery
Axons and Dendrites Branch off the main cell body Axons conduct impulses away from the cell body Dendrites convey impulses toward the cell body
Myelin Material that covers many axons and dendrites Enhances conduction along nerve fibers Gives the axons a white appearance; cell bodies without myelin are gray
Central Nervous System (CNS) Made up of the brain and spinal cord
Peripheral Nervous System Comprises all the nerves of the peripheral parts of the body, including spinal and cranial nerves
Brain Divided into the cerebrum, cerebellum, and brainstem
Cerebrospinal Fluid (CSF) Composed of water, glucose, sodium chloride, and protein Acts as a shock absorber for the brain and spinal cord
Spinal Cord Extends from the border of the first cervical vertebra (C1) to the level of the second lumbar vertebra (L2) Thirty-one pairs of spinal nerve roots exit the spinal cord, each consisting of a posterior sensory (afferent) root and anterior motor (efferent) root These roots, along with the 12 cranial nerves, make up the peripheral nervous system
Autonomic Nervous System Controls the involuntary activities of the viscera, including smooth muscle, cardiac muscle, and glands Two major subdivisions: sympathetic nervous system and the parasympathetic nervous system
Age-Related Changes The number of nerve cells decreases Brain weight is reduced; ventricles increase in size Lipofuscin: aging pigment deposited in nerve cells with amyloid, a type of protein Increased plaques and tangled fibers in nerve tissue Eye pupil often smaller; may respond to light more slowly Reflexes intact except for Achilles tendon jerk, which is often absent Reaction time increases, especially complex reactions Tremors in the head, face, and hands are common Some develop dizziness and problems with balance
Types of Disorders Developmental and genetic Trauma Infections and inflammation Neoplasms Degenerative processes Vascular disorders Metabolic and endocrine disorders
Nursing Assessment of Neurologic Function Health history Note speech, behavior, coordination, alertness, comprehension Chief complaint and history of present illness Document what prompted the patient to seek medical attention Describe any injuries If patient has pain, note the onset, severity, location, and duration Past medical history Head injury, seizures, diabetes mellitus, hypertension, heart disease, and cancer Record dates and types of immunizations
Nursing Assessment of Neurologic Function Family history Have immediate family members had heart disease, stroke, diabetes mellitus, cancer, seizure disorders, muscular dystrophy, or Huntington’s disease? Review of systems Fatigue or weakness, headache, dizziness, vertigo, changes in vision/hearing, tinnitus, drainage from ears or nose, dysphasia, neck pain or stiffness, vomiting, bladder or bowel function, sexual dysfunction, fainting, blackouts, tremors, paralysis, incoordination, numbness or tingling, memory problems, mood changes
Nursing Assessment of Neurologic Function Functional assessment Document whether present symptoms interfere with the patient’s usual activities and occupation Explore sources of stress, usual coping strategies, and sources of support Physical examination Level of consciousness, pupillary evaluation, neuromuscular response, and vital signs
Nursing Assessment of Neurologic Function Level of Consciousness: Anxious, agitated, combative – hyper-stimulated Somnolence – unnatural drowsiness or sleepiness Lethargy – like Somnolence, is excessive drowsiness Stupor – decreased responsiveness with a lack of spontaneous motor activity Semi-comatose – decreased responsiveness, no spontaneous motor activity (stuporous) but can be aroused usually with noxious or painful stimuli Comatose (Coma) – not responsive, not spontaneous motor activity and cannot be aroused with even painful stimuli
Diagnostic Tests and Procedures Advanced neurologic examination Cranial nerves Coordination and balance Neuromuscular function Sensory function Pain Temperature Light touch Vibration Position Tactile discrimination
BABINSKI REFLEX – B) negative Babinski – normal for adults C) positive Babinski – abnormal for adults Figure 27-7
Diagnostic Tests and Procedures Lumbar puncture Electroencephalography Electromyography Radiologic studies Brain scan Cerebral angiography and digital subtraction angiography Computed tomography Magnetic resonance imaging
Common Therapeutic Measures Drug therapy Antimicrobials Analgesics Anti-inflammatory Corticosteroids Anticonvulsants Diuretics Chemotherapeutic agents Dopaminergics Anticholinergics Cholinergics Antihistamines
Common Therapeutic Measures Surgery Craniotomy Surgical opening of the skull Craniectomy Excision of a segment of the skull Cranioplasty Any procedure done to repair a skull defect
Increased Intracranial Pressure (ICP) Normal ICP is 0-15 mmHg (averages 10-15) Brain, Blood, Cerebral Spinal Fluid are factors of ICP All must remain normal, if one factor increases ICP will rise; unless the other two factors have a decrease. When ICP increases, blood perfusion to brain decreases.
Assessment for Signs and Symptoms of Increasing ICP Level of consciousness (most reliable), pulpillary characteristics, motor function, sesnory function, vital signs. Abnormal motor function called posturing as ICP increases: Decorticate – abnormal flexation Decerebriate – abnormal extension (worse) Cushing’s Triad – Life threatening signs!
Assessment for Signs and Symptoms of Increasing ICP Cushing’s Triad - Hypertension with widening pulse pressure Irregular, shallow respirations Bradycardia
Treating Increasing ICP Positioning – Head of Bead no more than 30 Hyperventilation Fluid Management Mechanical drainage (ventriculostomy) Drug Therapy – Mannitol drug of choice, diuretic helps decrease ICP, increases UO
Migraine Headache Intracranial vasoconstriction followed by vasodilation Triggered by menstruation, ovulation, alcohol, some foods, stress Pain usually unilateral, often begins in the temple or eye area and is very intense Tearing and nausea and vomiting may occur Hypersensitive to light and sound; prefers dark, quiet environment Mild migraines treated with acetaminophen or aspirin; severe ones with ergotamine (Cafergot) or sumatriptan (Imitrex) tablet or auto injector for self-injection
Cluster Headache Occur in a series of episodes followed by a long period with no symptoms Intensely painful and seem to be related to stress or anxiety Usually have no warning symptoms Treatment may include cold application, indomethacin (Indocin), and tricyclic antidepressants
Tension Headache Result from prolonged muscle contraction from anxiety, stress, or stimuli from other sources, such as a brain tumor or an abscessed tooth Pain location may vary; may have nausea and vomiting, dizziness, tinnitus, or tearing Treatment: correction of known causes, psychotherapy, massage, heat application, and relaxation techniques Analgesics, usually nonopioid, may be prescribed to reduce anxiety
Seizure Disorder Electrical impulses in the brain are conducted in a highly chaotic pattern that yields abnormal activity and behavior Related to trauma, reduced cerebral perfusion, infection, electrolyte disturbances, poisoning, or tumors Medical diagnosis Accurate history of the seizure disorder Electroencephalogram (EEG)
Seizure Disorder Electrical impulses in the brain are conducted in a highly chaotic pattern that yields abnormal activity and behavior Related to trauma, reduced cerebral perfusion, infection, electrolyte disturbances, poisoning, or tumors Medical diagnosis Accurate history of the seizure disorder Electroencephalogram (EEG)
Seizure Disorder: Classification Partial seizures Simple Part of one cerebral hemisphere; consciousness not impaired Complex Consciousness impaired; may exhibit bizarre behavior Generalized seizures Involve the entire brain from the onset Consciousness lost during the ictal (seizure) period Types: tonic-clonic, absence, myoclonic, and atonic
Seizure Disorder: Phases of generalized tonic-clonic seizure (grand mal) Tonic phase Loss of consciousness, falling, crying and generalized stiffness. Clonic phase Jerking of the limbs Salivary frothing Postictal phase Deep sleep, extreme tiredness Headaches, confusion, irritability, vomitting
Seizure Disorder Status epilepticus Medical emergency: continuous seizures or repeated seizures in rapid succession for 30 minutes or more Aura Dizziness, numbness, visual or hearing disturbance, noting an offensive odor, or pain may precede a seizure minutes to seconds before a seizure. Medical treatment Resolution of the underlying condition Anticonvulsant drug therapy
Seizure Disorder Surgical treatment Removal of seizure foci in the temporal lobe and pallidotomy or vagal nerve stimulator
Seizure Disorder: Nursing Care Assessment Describe the seizure episode, including the postictal period (following the seizure), and document drug therapy
Seizure Disorder: Nursing Care Risk for Injury Side rails of bed up and padded, suction machine readily available, bed maintained in the low position Quickly move objects away from the patient Do not attempt to restrain the patient Ineffective Coping and Deficient Knowledge Teach family and patient about the seizure disorder and the therapy Teaching must be directed toward helping the patient and family adjust to a chronic condition Encourage questions and concerns