nrneph.2012.214-pt1

Nov 04, 2014

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Table 1 Evidence supporting the role of complement dysregulation and the benefit of C5 blockade in aHUS and C3G. Zuber, J. et al. (2012) Use of eculizumab for atypical haemolytic uraemic syndrome and C3 glomerulopathies Nat. Rev. Nephrol. doi:10.1038/nrneph.2012.214.

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eculizumab
complement dysregulation
c3 glomerulopathies
1038 nrneph
atypical haemolytic uraemic syndrome
table 1 evidence supporting

lucy-rowland

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nrneph.2012.214-pt1

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Table 1Evidence supporting the role of complement dysregulation and the benefit of C5 blockade in aHUS and C3G Zuber, J. et al.(2012) Use of eculizumab for atypical haemolytic uraemic syndrome and C3 glomerulopathies Nat. Rev. Nephrol.doi:10.1038/nrneph.2012.214

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Table 1 Risk of ipsilateral stroke after carotid endarterectomy compared with best medical therapy in NASCET and ECST at 5 years.

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Table 1 Studies in rats showing the involvement of vasopressin and V2Rs in CKDs, albuminuria and hypertension. Bankir, L. et al. (2013) Vasopressin: a novel target for the prevention and retardation of kidney disease? Nat. Rev. Nephrol. doi:10.1038/nrneph.2013.22.

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Figure 3 TLRs, NLRs and the NLRP3 inflammasome drive host defence and tissue repair mechanisms by the clearance of pathogens and cell debris and by regulation of repair pathways. Leemans, J. C. et al. (2014) Pattern recognition receptors and the inflammasome in kidney disease

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Table 1 Results from the Prostate Cancer Prevention Trial showing the numbers of men who underwent biopsy in each group and the numbers subsequently diagnosed with prostate cancer.

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nrneph.2012.214-pf2

Figure 2 Recovery of renal function is better with a shorter interval between onset of aHUS and initiation of eculizumab. Zuber, J. et al. (2012) Use of eculizumab for atypical haemolytic uraemic syndrome and C3 glomerulopathies Nat. Rev. Nephrol. doi:10.1038/nrneph.2012.214.

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Figure 1 Mechanisms by which aldosterone and/or MR activation induce oxidative stress, inflammation, and fibrosis. Brown, N. J. (2013) Contribution of aldosterone to cardiovascular and renal inflammation and fibrosis Nat. Rev. Nephrol. doi:10.1038/nrneph.2013.110.

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nrneph.2012.196-pf3

Figure 3 Comparison of a mild case and severe case of STEC-HUS treated at the University Hospital in Hamburg–Eppendorf during the German outbreak in 2011. Trachtman, H. et al . (2012) Renal and neurological involvement in typical Shiga toxin-associated HUS

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Figure 1 Renal survival of patients in our study with pauci-immune crescentic glomerulonephritis with and without ANCAs. Permission obtained from the American Society of Nephrology  Chen, M. et al. Antineutrophil cytoplasmic autoantibody negative pauci-immune crescentic glomerulonephritis.

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Table 1 Summary of a total of 72 cases of intracardiac leiomyomatosis reported between 1900 and 2005. Bennett ES et al. (2005) Intracardiac leiomyomatosis: iliac vein to right-ventricular outflow tract Nat Clin Pract Cardiovasc Med 2: 369 – 372 doi:10.1038/ ncpcardio0250.

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nrneph.2012.214-pt2

Table 2 Clinical and genetic characteristics of 24 off-trial patients treated with eculizumab for overt aHUS. Zuber, J. et al. (2012) Use of eculizumab for atypical haemolytic uraemic syndrome and C3 glomerulopathies Nat. Rev. Nephrol. doi:10.1038/nrneph.2012.214.

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Figure 2 Activation of C3 convertase is involved in both complement-mediated and immune-complex-mediated MPGN. Bomback, A. S. & Appel, G. B. (2012) Pathogenesis of the C3 glomerulopathies and reclassification of MPGN Nat. Rev. Nephrol. doi:10.1038/nrneph.2012.213.

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Peeling et al. Nature Reviews Microbiology 4 , 909 – 921 (December 2006) | doi:10.1038/nrmicro1555.

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