Figure 5

Oct 13, 2014

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Figure 5. Anna C. Seale. (2013) 74 , 00-00. doi:10.1038/pr.2013.207.

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Figure 5

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Figure 5 Anna C. Seale (2013) 74, 00-00. doi:10.1038/pr.2013.207

Figure 5-60

Figure 5-60. Page 122. EcoRI. Tetracycline Resistance. Ampicillin Resistance. PstI. HindIII. +. +. +. +. +. Origin of Replication. A plasmid. Figure 5-43 The pUC18 cloning vector. Page 106. Figure 5-46 Construction of a recombinant DNA molecule. Page 108.

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Figure 5-39

Figure 5-39. Analog to Analog Modulation.  The McGraw-Hill Companies, Inc., 1998. WCB/McGraw-Hill. Figure 5-40.  The McGraw-Hill Companies, Inc., 1998. WCB/McGraw-Hill. Figure 5-41. Amplitude Modulation.  The McGraw-Hill Companies, Inc., 1998. WCB/McGraw-Hill. Figure 5-42.

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Supplementary figure 5

A. GBT-B1 (^Gal4-VP16). B. GBT-B2 (^Gal4-FF). Supplementary Figure 1. D. Supplementary figure 5.

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Figure 5

Figure 1. Figure 4. Figure 3. Figure 5 Potential Growth from Establishing a MUHC PCP Relationship with Patients without a Designated PCP. Figure 2.

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Supplementary Figure 5

Supplementary Figure 5.

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Figure 5-22

Figure 5-22. Digital to Analog Encoding. 3 Characteristics of a sine wave Amplitude Frequency Phase 3 Mechanisms for Modulating Digital Data into Analog Signal ASK FSK PSK. Figure 5-23. Bit Rate and Baud Rate. Bit rate number of bits transmitted during 1 second Baud rate

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Supplemental Figure 5

Supplemental Figure 5. A. MTLn3 GFP-CXCL12. MTLn3 GFP-pQCXIP. C. B. ***. CXCR4. IgG. % of max. BMM Chemotaxis to CXCL12. % of max. CXCR4-PE. CXCR4-PE. IgG. CXCR4. B.

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Supplementary Figure 5.

Supplementary Figure 5. A. i.c.v. Ghrelin. AVV injection in VTA. FI. B. C. 2h. D.

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Supplemental Figure 5

Trastuzumab-treated. Control-treated. A. MDA-MB-453. 14.2%. 10.7%. DEAB-inhibited. DEAB-inhibited. B. JIMT-1. 5.2%. 5.4%. DEAB-inhibited. DEAB-inhibited. C. Sum159-HER2. 7.1%. 36%. DEAB-inhibited. DEAB-inhibited. Trastuzumab-treated. Control-treated. D. Supplemental Figure 5.

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Figure 5

2° ab alone. GPBAR1+ LPMC. Control TNBS + vehicle TNBS + Ciprofloxacin. A B E. Control TNBS TNBS

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Supplementary Figure 5

Supplementary Figure 5. A. B. MITOCHONDRIA. TOTAL. WT. S46A. WT. S46A. p53. RITA. –. +. +. –. WB: p53. % of apoptotic cells. α-amanitin. –. +. –. +. WB: VDAC. WB: HIPK2. WB: PCNA. WB: p53pS46. NT. RITA. WB: p53. WB: Actin. C. D. pLPC. Pin1-HA. MITOCHONDRIA. TOTAL.

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Supplemental Figure 5

Supplemental Figure 5.

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Suppl Figure 5

Suppl Figure 5. WT ctl1. WT ctl1. conditional defect. NO 3 - , Cl - , sucrose Hermans et al . (2010). Low, moderate NO 3 -. High NO 3 -. Endogenous content/production ABA ↓ C K ↑ C 2 H 4 ↑↑ Exogenous supply ABA induces the arm phenotype

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Figure 5-16

Figure 5-16. Analog to Digital Encoding.  The McGraw-Hill Companies, Inc., 1998. WCB/McGraw-Hill. Figure 5-17. PAM.  The McGraw-Hill Companies, Inc., 1998. WCB/McGraw-Hill. Figure 5-18. Quantized PAM Signal.  The McGraw-Hill Companies, Inc., 1998. WCB/McGraw-Hill. Figure 5-19.

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Supplemental Figure 5

Supplemental Figure 5. p <0.05. p =0.05. a. b. p <0.05. p <0.05. p <0.05. p <0.05. c. Muscle. d. Liver. + - + - UCP3. p <0.01. UCP3. SDH. p <0.05.

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Supplementary Figure 5

Supplementary Figure 5. A. 6h 12h 18h 24h. Rab 3B myosin heavy chain 7 putative cold shock domain protein A myosin heavy chain 4 myosin heavy chain 3 actinin alpha 2 E-cadherin. 0.2. B. 1.0. CONTROL 24 h. BaP 24 h. 5.0. (a). (b).

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Supplement Figure 5

(XB130). (XB130). Supplement Figure 5. A. B. C.

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Supplementary Figure 5

A. C. B. Supplementary Figure 5.

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Figure 5

Figure 5.

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Figure 5-15

Figure 5-15. Analog to Digital Encoding.  The McGraw-Hill Companies, Inc., 1998. WCB/McGraw-Hill. Figure 5-16. PAM.  The McGraw-Hill Companies, Inc., 1998. WCB/McGraw-Hill. Figure 5-17. Quantized PAM Signal.  The McGraw-Hill Companies, Inc., 1998. WCB/McGraw-Hill. Figure 5-18.

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Supplementary Figure 5

Supplementary Figure 5. a. T80. HEY. ATG5 shRNA H2-1 + FAC. ATG5 shRNA H2-2 + FAC. ATG5 shRNA H2-1 + FAC. ATG5 shRNA H2-2 + FAC. ATG5 shRNA H2-1. ATG5 shRNA H2-2. Control-1 + FAC. Control-2 + FAC. ATG5 shRNA H2-1. ATG5 shRNA H2-2. Control-1 + FAC. Control-2 + FAC. Control-1.

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Figure 5

b. a. Figure 5. c.

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