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IMAGING CONTRIBUTION IN ARACHNOID INTRACRANIAL CYST

IMAGING CONTRIBUTION IN ARACHNOID INTRACRANIAL CYST. E.GAMY-J.MAHLAOUI-T.AMIL-S-CHAOUIR-A.HANINE-M.MAHI-S.AKJOU J Medical imaging military hospital Mohammed V instruction –Rabat . NR3. Introduction .

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IMAGING CONTRIBUTION IN ARACHNOID INTRACRANIAL CYST

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  1. IMAGING CONTRIBUTION IN ARACHNOID INTRACRANIAL CYST E.GAMY-J.MAHLAOUI-T.AMIL-S-CHAOUIR-A.HANINE-M.MAHI-S.AKJOUJ Medical imaging military hospital Mohammed V instruction –Rabat. NR3

  2. Introduction • Intracranial arachnoid cysts are defined as a pocket full of intra-subarachnoid CSF without communication with the ventricular system. • The aim of this study is to clarify the contribution of computed tomography (CT) and especially MRI. • In the diagnosis with emphasis on information brought by the sequences (diffusion) in the differential diagnosis.

  3. Materials and Methods • CT scans performed in axial and coronal. • MRI includes the following morphological sequences weighted in T1, T2, FLAIR, and T2 * sequences RELEASE in the different planes.

  4. Results • CT shows a process of expansive cystic lesion that is hypodense and the same signal as cerebrospinal fluid (CSF), which can result in thinning of the cortex next, there is no contrast enhancement. • MRI it has a signal identical to that of (LCS) on the sequences T1 and T2 without contrast. However to make a difference with an epidermoid cyst, FLAIR-weighted sequences, distribution and CISS are a great contribution.

  5. CT: CSF density bone remodeling, no contrast enhancement.

  6. MRI: T1/T2: iso intense to CSF

  7. DWI: no signalno contrast enhancement

  8. DISCUSSION • There is no causal link between the temporal lobe hypoplasia and arachnoid cysts appear despite their association. • Hypothesis probable abnormalities of embryogenesis that affects • Independently, and the formation of the arachnoid, and the temporal lobe in some patients, is the effect of compression KA. • The search for evidence in favor of either MRI or hypogénésie compression of the temporal lobe by a KA.

  9. DISCUSSION • In The hypoplasia of the temporal lobe, temporal lobe concave next to the KA, • Discharge of the temporal horn and / or adjacent structures;sinuosity, ripple temporal cortex next to the KA. • Decrease in the volume of adjacent parenchyma. • Not discharge. • No thinning of cortical bone next to the KA.

  10. Differentialdiagnosis • Epidermoid cyst: • Irregular edge in <cauliflower>, is insunie in tanks, • Includes vessels and nerves • Registered in 45% of cases at the basal cisterns. •   Light Flair hyperintense signal and Hyper Distribution. • Light Flair hyperintense signal and Hyper Distribution

  11. Differentialdiagnosis • The chronic subdural hematoma: • Lenticular, higher signal to CSF​​ • Subdural hygroma • CAVITY porencephalic • MEGAGRANDE TANK • MALIGNANT CYSTIC • NEURO-CYSTS ENTERIC • CYST NEUROGLIAL

  12. TRAETMENT • KA asymptomatic abstention • KA giant symptomatic or asymptomatic high risk of bleeding:       - Craniotomy + resection of the outer mb

  13. Conclusion • The MRI allows the diagnosis of intracranial arachnoid cysts with characteristics of specific sequences that can differentiate epidermoid cysts. • With multi planar cuts it offers, it remains the best technique to assess the extent and anatomical relationships of these cysts.

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