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Osteopathic Considerations for the GI patient. UPPER MIDWEST OSTEOPATHIC HEALTH CONFERENCE May 2, 2014 Kate Heineman, DO and Shannon Crout DO. OMT for the GI patient:. Case Study Concept of nociception in osteopathic medicine Circulation Lymphatics Autonomics Respiration Mesenteries
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Osteopathic Considerations for the GI patient UPPER MIDWEST OSTEOPATHIC HEALTH CONFERENCE May 2, 2014 Kate Heineman, DO and Shannon Crout DO
OMT for the GI patient: • Case Study • Concept of nociception in osteopathic medicine • Circulation • Lymphatics • Autonomics • Respiration • Mesenteries • Lab
DDx: • Upper Abdominal Pain • Biliary colic • GERD • Peptic ulcer disease • Non-ulcer dyspepsia • Gastritis • Hiatal hernia • Cholecystitis • Cholangitis • Pancreatitis • Pneumonia • Myocardial infarction • Splenic abscess or infarction • Sub-diaphragmatic abscess • Hepatitis • Lower Abdominal Pain • Irritable bowel syndrome • Inflammatory bowel disease • Appendicitis • Diverticular disease • Kidney stones • Bladder distension • Pelvic pain • Diffuse Abdominal Pain • Mesenteric ischemia/infarction • Ruptured aneurysm • Abdominal wall pain • Gastroenteritis
OMT goals for the GI patient: • Relieve, improve, and enhance the patient’s abilities • To improve circulation • To improve visceral response to stress • To relieve congestion • To enhance removal of waste products from the tissues • To improve cardiac output • To improve oxygenation and nutrition at a cellular level • To enhance resistance to infection • To enhance predictable tissue levels and the pt’s response to medications • To enhance relaxation and comfort of the pt • Improve circulation, improve lymphatic flow, balance autonomic activity, improve respiration
OMT for the GI patient: • Case Study • Concept of nociception in osteopathic medicine • Circulation • Lymphatics • Autonomics • Respiration • Mesenteries • Lab
Case Study: BT • 51 y/o WF presents with biliary colic symptoms • HPI: 1-yr h/o intermittent postprandial RUQ pain, radiation of pain to mid-back and epigastric area after meals, worse with fatty foods, intermittent diarrhea and constipation • ROS: Denied weight loss, vomiting, hematochezia, dysuria, bowel or bladder incontinence • Previous evaluation by general surgeon: CMP was WNL, H. pylori negative, US of RUQ was negative for findings, US of pancreas was satisfactory, CCK-HIDA scan was negative with GB ejection fraction of 97%, biliary fluid was benign • Referral to gastroenterologist: EGD, biopsies and colonoscopy were negative for significant abnormalities
BT: • PMH: Hypothyroidism, seasonal allergies, h/o headaches • PSH: EGD, colonoscopy • Meds: levothyroxine, Topamax, Zyrtec D, Sudafed PRN, B-complex vitamin • Allergies: NKDA • Social Hx: Negative for tobacco, ETOH, illicit drug use • Family Hx: Negative for colon CA, celiac disease
Physical exam: • VS: BP 110/70, P 60, R 12 • General: 163-pound, healthy-appearing, WF • Neuro: CN II-XII grossly intact, +5/5 muscle strength testing for upper and lower extremities bilaterally, +2/4 DTR for all reflexes symmetric and bilateral, no noted motor or sensory deficits • Abdomen: Soft, NTTP, no rebound or guarding • Osteopathic structural exam: boggy tissue texture changes at T6-9 RrSl on the right with increased fascial drag, visceral pull toward GB, motion over anterior RUQ abdominal region of the sphincter of Oddi was palpated to have counterclockwise rotation, restriction of the superior 1/3 of the linea alba, sacrum was L on R BST, L5 FRSR
Assessment/Plan: • Working diagnosis: Biliary colic NOS, biliary dyskinesia • Treatment/plan: • OMT • ME to the thoracics and sacrum • BLT and MFR to the abdominal and lumbar regions • Response to treatment: • Pain in the epigastric region and back was improved • T6-9 somatic dysfunction was notably improved, although fascial drag was somewhat increased • Additional plan: • Magnesium supplementation 325 mg/day • Digestive enzymes one with each meal • Piston breathing for home exercise • F/U in 2 weeks
Follow up after 2 weeks: • RUQ pain had completely resolved • Bowels became much more regular • Compliant with supplement recommendation • Osteopathic structural findings: • Residual fascial drag T6-9 on right, no SB or R • Visceral pull to the GB much improved • Sphincter of Oddi had a clockwise rotation • L on L FST • Treatment/Plan: • OMT using BLT and MFR to the thoracics and abdomen and ME to the sacrum • Cont. digestive enzymes with meals for 3 months • Cont. the home piston breathing • F/U PRN
OMT for the GI patient: • Case Study • Concept of nociception in osteopathic medicine • Circulation • Lymphatics • Autonomics • Respiration • Mesenteries • Lab
The Concept of Nociception in Osteopathic Medicine http://www.angelfire.com/sc3/toxchick/medpharm/medpharm57.html
Spinal outflow resulting in palpatory somatic changes http://www.sciencedirect.com/science/article/pii/S0165017307000951
Primary Afferent Nociceptors (PANs) http://www.nature.com/nrn/journal/v5/n7/fig_tab/nrn1431_F1.html
Facilitation of the spinal cord by PANs http://www.studyblue.com/notes/note/n/cnspns/deck/3219889
Ward, RC. Foundations for Osteopathic Medicine, 2e, Lippincott Williams & Wilkins: 2002. Figure 8.2, pg. 139.
Sympathetic nerve supply of GB: T6-9 Ward, RC. Foundations for Osteopathic Medicine, 2e, Lippincott Williams & Wilkins: 2002. Figure 6.17, pg. 107
Allostasis: Ward, RC. Foundations for Osteopathic Medicine, 2e, Lippincott Williams & Wilkins: 2002. Figure 8.9, pg. 152
OMT for the GI patient: • Case Study • Concept of nociception in osteopathic medicine • Circulation • Lymphatics • Autonomics • Respiration • Mesenteries • Lab
“The rule of the artery is supreme.” • When blood and lymphatics flow freely, the tissues can perform their physiologic functions without impedance
Abdominal Aorta • Celiac a. • Left gastric a. • Splenic a. • short gastric arteries • splenic arteries • left gastroepiploic a. • Hepatic a. • cystic a. • R gastric a. • gastroduodenal a. • R gastroepiploic a. • superior pancreaticoduodenal a. • R hepatic a. • L hepatic a. • Superior mesenteric a. • jejunal and ileal arteries • inferior pancreaticoduodenal a. • middle colic a. • R colic a. • Ileocolic a • anterior cecal a. • posterior cecal a. – appendicular a. • ileal a. • colic a. • Inferior mesenteric a. • L colic a. • sigmoid arteries • superior rectal a.
Celiac Artery • Blood supply to: • Liver • Stomach • Abdominal esophagus • Spleen • Superior half of both the duodenum and the pancreas • Embryonic foregut
Superior Mesenteric Artery • Blood supply to • Cecum • Small intestine (except duodenum parts 1 and 2) • Ascending part of the colon • One-half of the transverse part of the colon • Embryonic midgut
Inferior Mesenteric Artery • Blood supply to • Second half of the transverse part of the colon • Descending colon, • Sigmoid colon • Rectum • Embryonic hindgut
OMT for the GI patient: • Case Study • Concept of nociception in osteopathic medicine • Circulation • Lymphatics • Autonomics • Respiration • Mesenteries • Lab
Lymphatics • Impaired lymph flow • Increased tissue congestion and impaired nutrient absorption from the bowel • Increased likelihood of fibrosis with increased scarring in the healing process. • Flow of lymph may be hindered by a poorly efficient, flattened diaphragm or by torsion of the fascia around the lymphatic channels located in the mesentery or at the thoracic inlet.
Cisterna chyli • The dilated portion of the thoracic duct at its origin in the lumbar region • Irregular fibromuscular sac the size of a cigarette (6 cm)
Treatment of lymphatics • Thoracic inlet • Re-dome thoracoabdominal diaphragm • Direct or indirect fascial treatment to the diaphragmatic attachments • Soft tissue treatment to the paraspinal muscles and quadratus lumborum • Pectoral traction • Pelvic diaphragm through the ischiorectal fossa. • Treat the lumbar, innominate, sacral regions to rebalance • Lymphatic pumps
OMT for the GI patient: • Case Study • Concept of nociception in osteopathic medicine • Circulation • Lymphatics • Autonomics • Respiration • Mesenteries • Lab
Parasympathetic dominance • Dominates innervation of the viscera during normal, long term, restful activity • Complaints of headaches, nausea, vomiting, diarrhea, cramps • Stimulation will increase the secretion rate of almost all gastrointestinal glands
Parasympathic considerations for the GI patient: CN X • PS innervation for the upper GI tract • Exits the skull thru jugular foramen
Parasympathetic dominance • Complaints of headaches, nausea, vomiting, diarrhea, cramps • Treat upper cervicals (OA, AA, C2) • Vagus nerve exits skull • Cranial • Vagus leaves cranium through the jugular foramen. • Suboccipital tension release • C3-5 somatic dysfunctions • Phrenic nerve to diaphragm • Sacrum, innominates, lumbosacral dysfunctions • Pelvic splancnicnerves • Sacral inhibition
Sympathetic considerations for the GI patient • The spinal cord becomes facilitated from the increased and prolonged visceral afferent input • Leads to palpatory tissue changes and tenderness to palpation in T5-11 (upper GI) or T9-L2 (lower GI) paraspinal muscles, the collateral ganglia, and Chapman’s reflex sites. • Preference for extension (small rotatores), rotation, and sidebending to the same side as the involved organ.
Sympathetic Dominance • Hyperactivity of the lower GI system is associated with • Ileus • Constipation • Abdominal distension • Flatulence
Chapman’s points • Stomach • Tender, palpable nodules on the anterior intercostal spaces between ribs 5/6 and 6/7 • Colon • Tender, palpable nodules on the lateral sides of the thighs in the anterior half of the iliotibial bands from the greater trochanters to the lateral epicondyles of the femurs
Rib Raising • The chain ganglia of the sympathetics lie in the fascia over the heads of the ribs • Applied to T5-T11 • Can be administered with the patient supine, lateral recumbent, or sitting • Position your finger pads at the rib angles • Wrists are placed onto the table such that a pressure can be applied through the shoulders and the elbows and into the wrists • The fingers are tractioned in a small amount in a lateral position. • Treatment only needs to be long enough to sense palpable tissue change (a few seconds to a few minutes) • Once a soft tissue release is appreciated, the hands are repositioned to subsequent ribs. • One should be able to treat approximately 5-6 ribs at one time.
Ventral abdominal inhibition • Celiac ganglia (T5-9) • Anterior to the abdominal aorta and between the xiphoid process and umbilicus • Separated into a R & L ganglion • Involved in upper GI disorders (stomach, duodenum, liver, GB, pancreas & spleen) • Superior mesenteric ganglion (T10-11) • Located around the base of the SMA • Innervates the entire small intestine below the duodenum, the R side of the colon, kidneys, adrenals, and gonads • Inferior mesenteric ganglion (T12-L2) • Located around the base of the IMA • Supplies the L colon and pelvic organs (except gonads)
Sympathetic dominance • Complaints of constipation, abdominal pain, flatulence, distention • Viscero-somatic reflexes • Chapman’s points • Rib raising • Sympathetic collateral ganglia inhibition (celiac, superior, inferior) • Sacral rocking • Stimulates parasympathetics
OMT for the GI patient: • Case Study • Concept of nociception in osteopathic medicine • Circulation • Lymphatics • Autonomics • Respiration • Mesenteries • Lab
Attending to mesenteries • Reduce congestion • Improve circulation • Free lymphatic pathways to the small intestines
Mesentery of the Small Intestine • Can be located in the pt by constructing an line 1 inch to the L and 1 inch above the umbilicus to a point in the RLQ just anterior to the R SI joint
OMT thoughts for the GI patient • Improve circulatory factors • Modify fascial patterns which hinder lymphatic patterns and pumps • Treat the base of the skull and upper cervical areas to affect parasympathetic function • Administer rib raising and paraspinal inhibition for autonomic imbalance and reflex dysfunction • OMT can help reduce the amount of pain medication required for patient’s comfort and can help prepare the patient’s body for better acceptance, distribution and utilization of specific medications