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A variety of completely different inflammatory models, sometimes experimentally induced colitis, arthritis, atherosclerosis, and respiratory tract inflammation, have been used. As much of this work has been carried out only inside the previous couple of years, massive-scale human trials are still lacking, although a few smaller studies on humans have proven some therapeutic promise. In the Diabetic intestine, it improves tight junctions and microflora. With C. difficile, it could decrease inflammation (IL-6).
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