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Bibliography of hypertrophy - Scalrovsky. 1. Fijensen en la izquierda muestra las presiones desarrolladas en ela ratas con hipertrofia, la hipertrofia disminuye la tension intramural ,mientras Que no dessarrollan hipertension hay una alta tension intramural. Ley de Laplace.
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Fijensen en la izquierda muestra las presiones desarrolladas en ela ratas con hipertrofia, la hipertrofia disminuye la tension intramural ,mientras Que no dessarrollan hipertension hay una alta tension intramural
Prepado de Langerdof en un raton , registra los potenciales de epicardio y endocardio y un globo intracavitaio r epi end Kel Kelly SHJ et al clonocall exp phasrmacilogy physiology 33.642;2006 Note que que el potencial de accion del endocardio se acorta cion el aumento de la pre4sion intracavitaria ,pero no hay cambios en el potencial de accion del epicardio
Molecular biology could explain the pattern of high voltage and fast conduction of R waves in left ventricle hypertrophy The high R wave in v4-5 in apical remodeling indicates the addition of sarcomeres in the longitudinal axis
Conclusions androgen receptors In cardiac myocytes from multiple species, including normal men and women,in a context that permits the cardiac phenotype and produce hypertrophy by direct receptors- specific mechanism for therapeutical or ilicit use of androgen in men
REModelacion tioca de un hipertenso Con remodelacion fibrotica sin hypertrofia muscular de alto riego para arritmias atriales y ventricularesל a)Una tipica remodelacionhypertrofia ventricular musculat
Spinal;e G Physiological Review 2007 Spinale F, Physiol Rev 2007
Metalloproteinases(MMP) regulate collagen I concentration in extra cellular matrix. The collagen turnover is about 90 days. Tissue Inhibitor Metalloproteinase (TIMP) regulates the concentration of MMP Lombardi R et al, Circulation 2003 Lombardi R et al, Circulation 2003
Physiol. Rev. 87: 1285-1342, 2007; doi:10.1152/physrev.00012.2007 0031-9333/07 $18.00 Physiol. Rev. 87: 1285-1342, 2007; doi:10.1152/physrev.00012.2007 0031-9333/07 $18.00 Physiol. Rev. 87: 1285-1342, 2007; doi:10.1152/physrev.00012.2007 0031-9333/07 $18.00 Physiol. Rev. 87: 1285-1342, 2007; doi:10.1152/physrev.00012.2007 0031-9333/07 $18.00 Myocardial Matrix Remodeling and the Matrix Metalloproteinases: Influence on Cardiac Form and Function Francis G. Spinale Myocardial Matrix Remodeling and the Matrix Metalloproteinases: Influence on Cardiac Form and Function Francis G. Spinale Division of Cardiothoracic Surgery, Medical Myocardial Matrix Remodeling and the Matrix Metalloproteinases: Influence on Cardiac Form and Function Francis G. Spinale Division of Cardiothoracic Surgery, Medical
. Part I: signal transduction and basic electrophysiological mechanisms Andreas Goette eurpace1,*and Uwe Lendeckel2 +Author Affiliations 1Division of Cardiology, University Hospital Magdeburg, Leipzigerstr. 44, Magdeburg 39120, Germany 2Institute of Experimental Internal Medicine, University Hospital Magdeburg, Magdeburg 39120, Germany *Corresponding author. Tel: +49 391 6713225; fax: +49 391 671 3202.E-mail address: andreas.goette@medizin.uni-magdeburg.de ReceivedOctober 17, 2007. AcceptedNovember 28, 2007. Next Section Abstract In the recent years, a tremendous amount has been learned about the pathophysiology and molecular biology of the cardiac angiotensin II system. Interestingly, the angiotensin II appears to have several effects on cardiac electrophysiology, which have not been fully appreciated so far. Therefore, this review will highlight on the basic knowledge of signal transduction and electrophysiological effects of angiotensin II, which may have an impact on the occurrence of cardiac arrhythmia. Key words Angiotensin Arrhythmias Fibrillati