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Understanding Muscle Contraction and Soreness: A Comprehensive Overview

Explore the types of muscle contractions, theories on muscle damage, effects of DOMS, and ways to analyze and recover from muscle soreness. Dive into the intricacies of muscle physiology and adaptation.

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Understanding Muscle Contraction and Soreness: A Comprehensive Overview

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  1. Concentric Contraction • Shortening of muscle • Force is generated • Train: increase strength

  2. Isometric Contraction • No change in length • Force is generated • Train: specific to joint angle

  3. Eccentric Contraction • Lengthening of muscle • Force is generated • Ability to generate most force • Utilizes less O2 than concentric • More hypertrophy and strength gains • More muscle damage

  4. Acute Muscle Soreness • Blood flow plays an important part • Build up of LA stimulates pain receptors • Soreness disappears when you stop activity

  5. Delayed Onset of Muscle Soreness (DOMS) • Eccentric work (pain peaks after 48 hours) • Isometric work (pain peaks after 12 hours) • LA disappears in one hour

  6. Eccentric contractions cause most damage • Could be due to: • Strains in the MU • Popping of sarcomeres • Stretching produces over-extended sarcomeres • May be that myosin heads are “loosely” bound during eccentric contractions and heads are “popped off” the chain, causing damage

  7. Lactic Acid Theory • Not likely • Why not?

  8. Spasm Theory • Spasm due to ischemia • Release of p substance that causes pain stimulus • Pain reflex: muscle continues to contract • Break spasm by: massage, ice, and stretching • p substance is unknown

  9. Connective Tissue Theory • 4 substances: LDH, 3mH (increases in urine), hydroxyproline (HP), and CPK • CPK and LDH leak into blood when muscle breaks down • HP increases in blood and urine when break down connective tissue • All four are markers of muscle damage

  10. DOMS: 5 components • Structural components of skeletal muscle and CT are disrupted • Structural damage causes a change in permeability in muscle cell, allowing high levels of calcium into cells (kills cells) • High calcium causes a release of proteolytic enzymes • Fragments of broken protein leak into serum/blood and lead to an immune response; WBC flood the area • Histamine response: fluid flows to area, swelling and heat production, stimulates pain receptors

  11. Can analyze damage via • MRI • Swelling • Muscle protein in blood • Impaired substrate utilization • Decrease in strength and ROM • Decrease in motor control

  12. If entire muscle is not damaged, small areas can recover • In resting state, z lines are zig-zagged • Connection between titin and myomesin is broken, distorting the myofibril arrangement • Desmin (connects z lines of different myofibrils) is gone 3 days post exercise • Fibronectin (connects membrane to matrix inside fiber) is also disrupted

  13. Most damage is repaired after 10 days • Soreness decreases with repeated bouts • Adaptation

  14. Use of NSAIDS and DOMS • Short term pain/repair • Actual • Placebo • Long term pain/repair

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