1 / 10

Final practicle

Final practicle. 3- Mucoepidermoid carcinoma: - is the most common primary malignant tumor - occurs mainly in parotid gland - tumor composed of mixture of neoplastic squamous and mucus-secreting cells. Familial polyposis syndrome. Autosomal dominant condition characterized by

Olivia
Download Presentation

Final practicle

An Image/Link below is provided (as is) to download presentation Download Policy: Content on the Website is provided to you AS IS for your information and personal use and may not be sold / licensed / shared on other websites without getting consent from its author. Content is provided to you AS IS for your information and personal use only. Download presentation by click this link. While downloading, if for some reason you are not able to download a presentation, the publisher may have deleted the file from their server. During download, if you can't get a presentation, the file might be deleted by the publisher.

E N D

Presentation Transcript

  1. Final practicle

  2. 3- Mucoepidermoid carcinoma: - is the most common primary malignant tumor - occurs mainly in parotid gland - tumor composed of mixture of neoplastic squamous and mucus-secreting cells

  3. Familial polyposis syndrome Autosomal dominant condition characterized by presence of multiple adenomatous polyps (500-2500) Polyps begin to develop between 10 and 20 years of age. All patients develop tubular adenomas and cancer*. Pathogenesis Inactivation of adenomatous polyposis coli (APC) suppressor gene* located on chromosome 5

  4. Acute viral hepatitis • ◆ What is the most likely diagnosis? • Acute viral hepatitis (hepatitis B virus) • ◆ What are the possible etiologies of this disorder? • Acute infection: Hepatitis B virus+Hepatitis A virus. • ◆ What other tests would be appropriate? • LFT, ELISA, PCR • ◆ What are the possible complications? • Progress to Chronic hepatitis or Fulminant hepatitis with confluent necrosis • ◆ Describe the morphologic features of this condition? • 1) Loss of normal architecture. 2) Diffuse Hepatocyte injury with ballooning degeneration. 3) Lobular Chronic inflammatory cells 4) Apoptosis-T-cell cytotoxic 5) Cholestasis. 6) interface hepatitis. 7) Regenerating hepatocytes are large, frequently containing multiple nuclei

  5. - What is the diagnosis?- Write any two etiological agents?- Write tow morphological features to support the diagnosis?

  6. Diagnosis: Fulminant hepatitis • Etiological agents: • Infectious causes: • Viral hepatitis is responsible for about 12% of cases • 8% are caused by HBV infection and the rest by HAV& HCV. • Herpesvirus infection. • Dengue virus. 2. Non-infectious causes • Drug- Acetaminophen toxicity. 3. Unknown. • Grossly- • The capsule is wrinkled. • Microscopic features: • Portal tracts and terminal hepatic veins are closer together than normal. • Massive necrosis with collapse of the intervening parenchyma. • Massive influx of macrophages for phagocytosis. • Ductular regeneration. • An infiltrate of mononuclear inflammatory cells is present

  7. Lab diagnosis • Diagnosis through detection of characteristic eggs & proglottids in stool wet mount • Detection of antibodies against the parasite in the patients serum by serological tests like ELISA, CDC immunoblast assay • Neuroimmaging like CT scan and MRI • Treatment • Praziquantal or niclosamide • - Surgery for neurocysticercosis

  8. البايو كيم كان كيف والجواب كان للسؤال الأول :obstructive joundiceالثانيabsence of urobilogen & strictobilogen

  9. الاناتوميجاflasiformligamintquadrate lobeوالستيشن الثانيuterussoas muscle

More Related