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Lecture 9.3 rad240 pathology

Lecture 9.3 rad240 pathology. Dr shai’. POLYPS. ANY mucosal bulging, blebbing, or bump HYPERPLASTIC (NON-NEOPLASTIC) HAMARTOMATOUS (NON-NEOPLASTIC) ADENOMATOUS (TRUE NEOPLASM, and regarded by many as “potentially” PRE-MALIGNANT as well) SESSILE vs. PEDUNCULATED TUBULAR vs. VILLOUS.

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Lecture 9.3 rad240 pathology

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  1. Lecture 9.3rad240 pathology Dr shai’

  2. POLYPS • ANY mucosal bulging, blebbing, or bump • HYPERPLASTIC (NON-NEOPLASTIC) • HAMARTOMATOUS (NON-NEOPLASTIC) • ADENOMATOUS (TRUE NEOPLASM, and regarded by many as “potentially” PRE-MALIGNANT as well) • SESSILE vs. PEDUNCULATED • TUBULAR vs. VILLOUS

  3. POLYPS

  4. PEDUNCULATED vs VILLOUS vs SESSILE

  5. BENIGN vs. MALIGNANT • Usual, atypia, pleo-, hyper-, mitoses, etc. • Stalk invasion!!!

  6. HPERPLASTIC POLYP

  7. ADENOMATOUS POLYP (TUBULAR)

  8. ADENOMATOUS POLYP (VILLOUS)

  9. “FAMILIAL” NEOPLASMS • 1) POLYPOSIS (NON-NEOPLASTIC, hamartomatous) • 2) POLYPOSIS (NEOPLASTIC, i.e., cancer risk) • 3) HNPCC: (Hereditary Non Polyposis Colorectal Cancer)

  10. MUCINOUS CYSTADENO(CARCINO)MA ADENOMA CARCINOMA

  11. PERITONEUM • Visceral, Parietal: all lined by mesothelium • Peritonitis, acute: • Appendicitis, local or with rupture • Peptic ulcer, local or ruptured • Cholecystitis, local or ruptured • Diverticulitis, local or with rupture • Salpingitis gonococcal or chlamydial, retrograde or perforated • Ruptured bowel due to any reason • Perforating abdominal wall injuries

  12. PERITONITIS • E. coli • STREP • S. aureus • ENTEROCOCCUS

  13. PERITONITIS, outcomes: • Complete RESOLUTION • Walled off ABSCESS • ADHESIONS

  14. SCLEROSING RETROPERITONITIS • Unknown cause (autoimmune?) • Generalized retroperitoneal fibrosis, progressive hydronephrosis

  15. TUMORS • MESOTHELIOMAS (solitary nodules or diffuse constricting growth pattern, also asbestos caused) • METASTATIC, usually diffuse, often looking very much like pseudomyxoma peritoneii, but containing tumor cells, usually adenocarcinoma

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