Module 5 Pediatric Cardiac Disorders

1. Module 5 Pediatric Cardiac Disorders

2. Fetal Circulation What is the main route of blood flow through the fetal heart?

3. Fetal Circulation Why does the blood flow in that route?

4. Fetal Circulation What is the stimulus for the change in circulation?

5. Intrauterine to Extrauterine What Happens after the Umbilical cord is Clamped?

6. Oxygen Saturation • What is oxygen saturation? • What is normal oxygen saturation levels? • What values indicate hypoxemia? • Why is it important for the nurse to know the oxygen saturation levels? question 5

7. Congestive Heart Failure

8. Congestive Heart Failure • What is wrong with the heart?

9. Congestive heart failure • What is the effect on: • Heart rate Preload Contractitility Afterload

10. Congestive Heart Failure • Why does the pump fail?

11. Etiology and Pathophysiology

12. Congestive Heart Failure • What does the body do to compensate for this congestion and heart failure?

13. Compensatory Mechanisms • With a decrease in Cardiac Output • Stimulation of the sympathetic nervous system • Tachycardia - increases venous return to the heart which stretches the myocardial fibers and increases preload.

14. Compensatory Mechanisms • With a decrease in cardiac output • Decrease perfusion to the kidneys and glomerulus • Increased renin and ADH secretion • Increase in Na and H2O retention to increase intravascular volume

15. Early Signs of CHF • The earliest signs are often subtle: • Infant will have mild resting tachypnea • Increasing difficulty feeding A Change in Condition

16. Signs and Symptoms • Pulmonary congestion • Tires easily during feeding • Tachypnea, Dyspnea, orthopnea • Signs of respiratory distress • Wheezing, rales and rhonchi • Easily fatigue • Impaired cardiac output • Tachycardia • Extremities cool, capillary refill >2 seconds • Diaphoretic, sweating, hypotension

17. Signs and Symptoms • Systemic venous congestion • Hepatomegaly • Edema • Weight gain • High metabolic rate • Failure to thrive • Slow weight gain

18. Goal of Treatment

19. Treatment of Congestive Heart Failure • Medication Therapy • Digitalis – increases contractility and decreases heart rate. • ACE-inhibitors - blocks release of angiotension-aldosterone; arterial vasodilator / afterload reducing agent • Diuretics - enhance renal secretion of sodium and water by reducing circulating blood volume and decreasing preload,  pulmonary congestion. • Beta Blocker - increases contractility

20. Nursing Care How would the nurse recognize digitalis toxicity in an infant or child? What are the pulse rate criteria in administration of digitalis?

21. Digitalis • Digitalization • Given in divided doses • Maintenance • Given daily, usually in two divided doses • Therapeutic vs. Toxicity • Therapeutic range – 0.8 to 2.0 ng/ml • Toxicity • **EKG changes – arrhythmia • Slow pulse- bradycardia • Vomiting – very rare in infants

22. Digitalis • Why are we so concerned with the potassium levels when the child is on digitalis therapy?

23. Treatment of Congestive Heart Failure • What is the type of Diet most commonly ordered? • How would nursing measure are used to decrease stress on the heart?

24. Feeding the child with CHF • Feed the infant or child in a relaxed environment; frequent, small feedings may be less tiring • Hold infant in upright position; may provide less stomach compression and improve respiratory effort • If child unable to consume appropriate amount during 30-minute feeding q 3 h, consider nasogastric feeding • Monitor for increased tachypnea, diaphoresis, or feeding intolerance (vomiting) • Concentrating formula to 27 kcal/oz may increase caloric intake without increasing infant’s work

25. Cardiac Catheterization Measure oxygen saturations and pressure in the cardiac chambers and great arteries Evaluate cardiac output

26. Cardiac Catheterization • This process involves passing a catheter through the femoral vein or artery into the heart. • Performed to evaluate heart valves, heart function and blood supply, or heart abnormalities in newborns.

27. Cardiac Catheterization • Pre-care: • History and Physical • Lab work – EKG, ECHO cardiogram, CBC • NPO • Vital signs • Preprocedural teaching

28. Best Nursing Action • During post procedure assessment, the nurse notes bleeding at the insertion site. • What should the nurse do first? • What additional interventions are implemented?

29. Post Cardiac Catheterization Care What is Wrong with this picture?

30. Post Cardiac Catheterization Care Mother Reading to child

31. Congenital Cardiac Anomalies

32. Ask Yourself? • What is the most common assessment finding indicating a cardiac anomaly? • Answer: an audible heart murmur

33. Left-to-Right Shunting Defects Patent Ductus Arterious Atrial septal defects Ventricle septal defects

34. Atrial Septal Defect • Oxygenated blood is shunted from left to right side of the heart via defect • A larger volume of blood than normal must be handled by the right side of the heart hypertrophy • Extra blood then passes through the pulmonary artery into the lungs, causing higher pressure than normal in the blood vessels in the lungs  congestive heart failure

35. Treatment • Medical Management • Medications – digoxin • Cardiac Catheterizaton - • Amplatzer septal occluder • Open-heart Surgery

36. Treatment • Device Closure – Amplatzer septal occluder During cardiac catheterization the occluder is placed in the Defect

37. Ventricle Septal Defect • Oxygenated blood is shunted from left to right side of the heart via defect • A larger volume of blood than normal must be handled by the right side of the heart hypertrophy • Extra blood then passes through the pulmonary artery into the lungs, causing higher pressure than normal in the blood vessels in the lungs  congestive heart failure

38. Treatment Surgical repair with a patch inserted

39. Patent Ductus Arteriosus • Blood shunts from aorta (left) to the pulmonary artery (right) • Returns to the lungs causing increase pressure in the lung • Congestive heart failure

40. Treatment for PDA • Medical Management • Medication • Indomethacin - inhibits prostaglandin's . (When levels of prostaglandins are decreased, the ductus closes) • Surgery • Ligate the • ductus arteriosus

41. Treatment for PDA • Cardiac Catheterization • Insert coil – tiny fibers occlude the ductus arteriosus when a thrombus forms in the mass of fabric and wire

42. Cardiac Anomalies - Treatment When is surgery Performed? Why?

43. Obstructive or Stenotic Lesions Pulmonic stenosis coarctation of aorta

44. Pulmonic Stenosis • Narrowing of entrance that decreases blood flow • Increases preload causes right ventricular hypertrophy

45. Obstructive or Stenotic Lesions • Treatment: • Medications – Prostaglandins to keep the PDA open • Cardiac Catheterization • Baloon Valvuloplasty • Surgery • Valvotomy

46. Aortic Stenosis • The aortic valve is thickened and rigid • Stenosis creates left ventricular hypertrophy • Left ventricle may not be large enough to eject a normal cardiac output.

47. Aortic Stenosis • Symptoms • Poor peripheral perfusion, feeding difficulties, CHF • Treatment • Balloon valvoplasty • Surgery

48. Coarctation of the Aorta • Narrowing of Aorta causing obstruction of left ventricular blood flow • Left ventricular hypertrophy

49. Signs and Symptoms • What are B/P findings support the diagnosis? • What is different in the pulses? • Why would the patient C/O leg pains? • What causes nose bleeds?

50. Treatment • Goals of management are to improve ventricular function and restore blood flow to the lower body. • Medical management with Medication • A continuous intravenous medication, prostaglandin (PGE-1), is used to open the ductus arteriosus allowing blood flow to areas beyond the coarctation. • Baloon Valvoplasty