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MUSCLE ENERGY TECHNIQUES (MET)

MUSCLE ENERGY TECHNIQUES (MET). By:- Dr. Hardik D. Patel PT, MIAP. What Is Manual Therapy…??.

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MUSCLE ENERGY TECHNIQUES (MET)

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  1. MUSCLE ENERGY TECHNIQUES(MET) By:- Dr. Hardik D. Patel PT, MIAP

  2. What Is Manual Therapy…??

  3. A great revolution has taken place in manipulative therapy , which involves a movement which is different from highvelocity/low amplitude thrusts and it is directed towards gentler methods which mainly involves soft tissue component • Different osteopathic physicians and physical therapistsdescribes all these techniques in its own way....

  4. MET is emerged from the osteopathic tradition • Mainly evolution of treatment methods, like involving isometric contraction and stretching, used in physical therapy, called PNF • PNF method tended to stress the importance of rotational components • Usually involving extremelystrong contractions

  5. Initially, the focus of PNF related to the strengthening of neurologically weakened muscles, with attention to the release of muscle spasticityfollowing on from this, as well as to improving range of motion at intervertebral levels • But Fred Mitchell snr. adapted this technique for use in joint mobilization and release of muscle shortness, and it was a natural evolution which has continued in physiotherapy, manual medicine, osteopathy and right now increasingly in massage therapy and chiropractic settings

  6. Two forms of MET • Post Isometric Relaxation (PIR) and • Reciprocal Inhibition(RI)

  7. Post Isometric Relaxation The term post isometric relaxation (PIR) refers to the effect of the subsequent reduction in tone experienced by a muscle, or group of muscles, after brief periods during which an isometric contraction has been performed

  8. neurological effects of the loading of the Golgi tendon organs of a skeletal muscle by means of an isometric contraction, which produce a postisometric relaxation effect in that muscle

  9. Reciprocal Inhibition • When a muscle is isometrically contracted, its antagonist will be inhibited, and will demonstrate reduced tone immediately following this • Thus the antagonist of a shortened muscle, or group of muscles, may be isometrically contracted in order to achieve a degree of ease and additional movement potential in the shortened tissues

  10. reciprocal effect of an isometric contraction of a skeletal muscle, resulting in an inhibitory influence on its antagonist

  11. Variations on the MET theme 1. Lewit’s postisometric relaxation method 2. Janda’s postfacilitation stretch method 3. Reciprocal inhibition variation 4. Strengthening variation

  12. Lewit’s postisometric relaxation method • The hypertonic muscle is taken, and find barrier point where resistance to movement is first noted • Isometrically contracts the affected hypertonic muscle away from the barrier (agonistis used) • The degree of effort is very minimal, say 20% of his available strength • Patient is asked to exhale and relax completely • Muscle is taken to a new barrier with all slack removed but no stretch • Starting from this new barrier, the procedure is repeated two or three times

  13. What is happening? • During resistance using minimal force (isometric contraction) only a very few fibers are active, the others being inhibited • During relaxation (in which the shortened musculature is taken gently to its new limit without stretching) the stretch reflex is avoided – a reflex which may be brought about even by passive and non-painful stretch

  14. Note:- • According to Lewit, Stretching of muscles during MET, is only required when contracture due to fibrotic change has occurred • Lewit suggests that trigger points and ‘fibrositic’ changes in muscle will often disappear after MET contraction methods • He suggests that, also referred local pain points, resulting from problems elsewhere, will also disappear more effectively than where local anaesthesia or needling (acupuncture) methods are employed

  15. Janda’s postfacilitation stretch method Stronger isometric contraction than that suggested by Lewit • The shortened muscle placed in a mid-range position • Contracts the muscle isometrically, using a maximum degree of effort for 5–10 seconds while the effort is resisted completely • On release of the effort, a rapid stretch is made to a new barrier, without any ‘bounce’, and this is held for at least 10 seconds • The patient relaxes for approximately 20 seconds and the procedure is repeated between three and five times more

  16. Reciprocal inhibition variation • Mainly used in acute settings, where tissue damage or pain precludes the use of the usual agonist contraction (use of antagonist) • Muscle is placed in a mid-range position • Isometric or Isotonic muscle contraction • On ceasing the effort, the patient inhales and exhales fully, at which time the muscle is passively lengthened

  17. Strengthening variation • Also called isokinetic contraction (also known as progressive resisted exercise) • Patient starts with a weak effort but rapidly progresses to a maximal contraction of the affected muscle(s) • Find area of weakness during full movement range • Strengthening of weak musculature in areas of permanent limitation of mobility is seen as an important contribution in which isokinetic contractions may assist

  18. Patterns of function and dysfunction • Why do soft tissues change from their normal elastic, pliable, adequately toned functional status to become short, contracted, fibrosed, weak, lengthened and/or painful………..?????

  19. The reasons are many and varied • Usually compound, and may be summarised under broad headings such as biomechanical, biochemical and psychological • Or under more pointed headings such as overuse, abuse, misuse, disuse • Time related - acute, subacute or chronic

  20. In order to make sense of what is happening when a patient presents with symptoms, it is necessary to be able to extract information from the patient about his condition

  21. Information made available via observation, palpation and examination • Postural (structural) • Motion (functional) • Postural muscle grid • Muscular weakness grid • Fascial patterns • Local dysfunction • Breathing function (and dysfunction)

  22. THE EVOLUTION OF MUSCULOSKELETAL DYSFUNCTION • Acquired postural imbalances • stress • Inborn imbalance • The effects of hyper- or hypo mobile joints, including arthritic changes • Trauma, inflammation and subsequent fibrosis • Disuse, immobilization • Reflexogenic influences (viscerosomatic, myofascial and other reflex inputs) • Climatic stress such as chilling • Nutritional imbalances (vitamin C deficiency reduces collagen efficiency) • Infection

  23. Deterioration of muscle function can be demonstrated by three syndromes • Hypotonia which can be determined by inspection and palpation • Decrease in strength which can be determined by MMT • Changed sequence of activation in principal movement patterns, which can be more easily observed and evaluated

  24. Characteristics of altered movement patterns • The start of a muscle’s activation is delayed, resulting in an alteration in muscles activation sequence • Overall decrease in activity in the affected muscle • In extreme cases, EMG readings shows completely silent. This can lead to a misinterpretation that muscle strength is totally lacking when in fact, after proper facilitation, it may be capable of being activated towards more normal function. (Janda calls these changes ‘pseudoparesis’.) • Non-inhibited synergists or stabilizers often activate earlier

  25. Mainly two types of muscles, we have to consider when we are talking about MET

  26. Postural / phasic muscle characteristics

  27. Postural muscles that shorten under stress

  28. PATTERNS OF DYSFUNCTION

  29. Upper Crossed Syndrome

  30. Lower Crossed Syndrome

  31. Fibromyalgia and trigger points

  32. Temporalis, Masseter, SCM, Splenius Capitis, Upper & Lower Trepezius, Levator Scapulae, Post. Cervical

  33. ECR, Supinator, Multifidus, Gluteus Medius, Scaleni, Infraspinatus, Supraspinatus

  34. Pectoralis Major, Serratus Ant., Deltoid, Tibialis Ant., Gastrocnemius, Soleus, Peroneus Longus

  35. Gluteus Minimus, Vastus Medialis, Biceps Femoris, Adductor Longus

  36. Eyes can not see what mind doesn't know

  37. How to use MET……..??? • PALPATION SKILLS • IDENTIFY BASIC BARRIER • MET USING IN……  ACUTE CONDITIONS  CHRONIC CONDITIONS

  38. COMMON ERRORS DURING APPLICATION

  39. Patient`s errors during MET • Contraction is too hard • Contraction is in the wrong direction • Contraction is not sustained for long enough • Does not relax completely after the contraction

  40. Practitioner's errors in application of MET • Inadequate patient instruction is given • Inaccurate control of position of joint or muscle in relation to the resistance barrier • Inadequate counterforce to the contraction

  41. Counterforce is applied in an inappropriate direction • Moving to a new position too hastily after the contraction • The practitioner fails to maintain the stretch position for a period of time which allows connective tissue to begin to lengthen

  42. Contraindications And Side Effects Of MET • If any pathology is suspected, no MET should be used until an accurate diagnosis has been established • According to pathology, dosage of application can be modified accordingly like…..amount of effort used, number of repetitions, stretching introduced or not, etc

  43. As to side-effects, Greenman explains……… muscle contractions ↓ influence surrounding fascia, connective tissue ground substance and interstitial fluids ↓ influences its biomechanical function, but also its biochemical and immunological functions ↓

  44. muscle effort requires energy by the metabolic process ↓ results in carbon dioxide, lactic acid and other metabolic waste products which must be transported and metabolized ↓ reason that the patient will frequently experience some increase in muscle soreness within the first 12 to 36 hours following MET treatment

  45. If beginners to MET, then…. • Follow statement “cause no pain when using MET “ • Stick to light (20% of strength) contractions • Do not stretch over-enthusiastically • Have the patient's assistance in stretch

  46. Sequential assessment and MET treatment of main postural muscles Mainly musculoskeletal system pain ↓ related to muscle shortening ↓ eg. When any muscle is weak (reduction in tone) ↓ antagonists of these weak muscles get shortened (reciprocal inhibited tone)

  47. So………….. Prior to any effort to strengthen weak muscle ↓ shortened one should be dealt with appropriate means ↓ then toning of weak muscle is emphasized

  48. mostly seen that…….. tight muscles never loose its tone ( maintain their strength ) ↓ which leads to rapid recovery, when toning is emphasized

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