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Cardiology II. Arrhythmias. Objectives. Describe the normal conduction in the heart Describe pathophysiology of bradycardias Describe pathophysiology of tachycardias Describe treatment of the above. Case.
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Cardiology II Arrhythmias
Objectives • Describe the normal conduction in the heart • Describe pathophysiology of bradycardias • Describe pathophysiology of tachycardias • Describe treatment of the above
Case • A 55 year old male calls 911 because his “heart is racing.” He initially is alert, oriented, and has mild shortness of breath. On physical exam, he has a regular tachycardia at 180, and monitor shows a regular, narrow-complex tachycardia. He denies chest pain. Midway through transport, he becomes less responsive, and his blood pressure drops as he starts sweating profusely.
Bradycardias • Caused by damage to the conduction system resulting in a “broken road” or by drugs (beta blocker, calcium channel blockers) • If due to drugs, reversible. If not, many require a permanent pacemaker • Our goal is to temporarily support the heart rate until one of those two happen. • Atropine or External Pacing are options.
Branch Blocks • “Hiccups” on the conduction highway past the AV node • Can involve the right bundle, the left bundle, or divisions of the left bundle
Tachycardias • Any heart rate greater than 100. • The key is using the pattern to determine what is causing the tachycardia • The treatment is even more diverse, thus, knowing the pattern is critical to choosing the correct therapy. • The three main branches of tachycardia are atrial, nodal, and ventricular
Treatment Summary • EMT • ABCs • Call for ALS • Rapid Transport • AEMT • IV • Cardioversion of V-tach, V-fib • Paramedic • Atropine, pacing for bradycardias • Vagal Maneuvers, Adenosine for narrow complex tachyarrhythmias • Amio, cardioversion for wide complex tachyarrhythmia