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Epidemiology. 0.5-1% of UK population suffer burn /year.10% of these require admissionOf these admissions, 10% are life threatening75,000 Burn victims admitted to hospital every year in USA45% of US admissions for the scald burn are in the children < 5years of ageFlame burn are the largest grou
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1. BURNS Dr. Eyad Baqain
Plastic and Reconstructive Surgery
2. Epidemiology 0.5-1% of UK population suffer burn /year.
10% of these require admission
Of these admissions, 10% are life threatening
75,000 Burn victims admitted to hospital every year in USA
45% of US admissions for the scald burn are in the children < 5years of age
Flame burn are the largest group of patients admitted to a burn unit
3. High Risk Groups The very young
The very old
The very unlucky (21% are bystanders)
The very careless ( ¾th from there own action)
4. THE VERY YOUNG
5. THE UNLUCKY
6. THE VERY CARELESS
7. TYPES OF BURNS Flame burns; fires
Scold burns; hot liquids
Electrical burns; lightening injuries
Chemical burns; acids & alkali
8. FLAME BURN
9. SCOLD BURN
10. CHEMICAL BURN
11. CHEMICAL BURN
12. ELECTRICAL BURN
13. Burn wound ? Cell damage starts at 41 Deg Celsius
Coagulation of Protein > 50 Deg Celsius
Depth of wound: Temperature, Duration
Chemicals: ph, strength
Electricity: voltage, entry-exit points
14. Indications for admission to BU > 5% FT at any age
> 10% PT in <10 yrs >50yrs
> 20% PT in adults ,between 10 and 50 yrs
Special areas: face, perineum, hands, feet
Electrical ( including lightening)
Chemical
Inhalational injury
15. Indications for admission to BU Burns in patients with medical disorders
Burns in patients with concomitant trauma
Circumferential burns
Suspected abuse
Extremes of age
16. Organization of Burn Care
17. First Aid scene Extinguish flame, switch off power source, remove chemical soaked clothes, etc
Cool burn wound
Dilute acid- alkali
18. First Aid Scene Cool burn wound:
- reduce direct thermal trauma & stabilize mast cells, reducing release of histamine and other inflammatory mediators
- pain relief
- running water (15 degree C.)
- worth considering for up to 2 hours
19. Treatment in the A&E A B C, Like all other trauma patients, should be evaluated systematically
Intubation ??
Look for other injuries
Assess burn wound
20. Intubation :
Suspicion of inhalational injury
Severe facial & neck burn
easy early intubation will become difficult and impossible later on
21. Inhalational injury Fires
Closed space
Singed nasal hair
Carbonaceous material in nose & mouth
22. INHALATIONAL INJURY
23. Inhalational injury Direct thermal injury (upper airway)
Chemical injury ; products of combustion (lower airway)
CO poisoning ; systemic effect
24. CO poisoning Toxic symptoms > 20%, death at 60%
CO-Hb > 5% is indicative of inhalational injury but not severity
200 250 × greater affinity for Hb than oxygen
Treatment ; 100% O2, hyperbaric oxygen
25. Assessment of burn wound Remove all clothes/ maintain warm temp.
Remove all jewellery
Check the back
Estimate % BSA burn
Estimate depth
Recognize need for escharotomy
27. Large Burns (20 - 30%) Quantity of mediators is large : whole body oedema
Hypovolaemic shock
Fall in plasma volume
Fall in cardiac output
28. Management of Major Burns Initial patient management
Intravenous access established & I.V. fluids
N/G tube ; ileus, enteral feeding
Folleys catheter
Analgesia
Tetanus vaccine
Patient must be kept warm
Dressing
29. Intravenous fluids Parkland formula :
4 ml R.L. × %TBSA × Wt (kg)
half over 8 hrs
half over 16 hrs
0.5 ml /kg /%TBSA of 5% albumin in RL 24 hrs after injury , over 8 hrs ( for > 30% burn)
Children : 3ml R.L.× %TBSA × Wt
+ maintainance (G/S 0.45%)
30.
Formula is a guideline
U.O.P. of 0.5 ml/kg/hr in adults
U.O.P. of 1 ml/kg/hr in children
31. Extra Fluid is Required in Children
Inhalation injury
Electrical injury
Delayed resuscitation
Dehydration
Fire-fighters
Intoxicated patients
32. Differences in Children Limited physiologic reserve
Tendency to Hypoglycemia
Greater surface area to mass ratio
IV fluid required at a lower %TBSA
Need higher volume per kilogram
33. Monitoring Resuscitation Urinary output
Heart rate
Blood pressure
Central invasive haemodynamic
Electrolytes
Blood gases
pH (<7.39 - lactic acidosis ?)
34. Burn wound depth Superficial (first degree)
Partial thickness (second degree)
superficial partial
deep partial
Full thickness (third degree)
35. Superficial (epidermal) Burn Redness
Painful
Dry
Spontaneous scarless healing within 7 days
38. Partial Thicknesssuperficial Blister
Redness
Moist
Painful
Oedema
Reepithelialisation within 14 days with minimal to no scarring
41. Partial thicknessdeep Grey-white
Dry
Little to no pain
Reepithelialization with copious scarring over weeks (extensive collagen deposition)
43. Full thickness Dry
No pain
Clay like colour, white, charred
Leathery
No spontaneous wound closure possible
(except very small wounds)
48. Burn Wound Management
49. Maximise Function and Appearance 1. Function - early reconstruction
2. Appearance - early reconstruction
(particularly face)
50. FINGERS CONTRACTURE!!!!!
51. BURN DEFORMITY
56. FULL THICKNESS BURN
57. SKIN GRAFT
58. SIX MONTHS LATER
59. SIX MONTHS LATER
60. Functions of the Skin Protects against bacteria
Prevents fluid loss
Regulates temperature
Initiates immune response
Sensation
Aesthetic & psychological importance
61. Escharotomy
Chest: To allow respiratory movement
Limb: To restore circulation in limb with excess swelling under rigid eschar
62. Limbs: Signs of Circulatory Obstruction Loss of distal circulation
pallor
coolness
absent pulse
loss capillary refill
decreased oxygen saturation
Pain on passive extension
Deep pain at rest
64. Estimation of % TBSA burn
65. Rules of NinesAdults
66. Rules of NinesChildren
67. Lund and Browder Chart
69. OUTCOME PROGNOSIS
Sum of Age in years
Plus
Area of burn in % TBSA
< 80 good
80-100 life threatening
>100 bad
70. OUTCOME In 1993 the LA 50 ( defined as burn size lethal to 50% of patients) in most burn units approached 75% total body surface area .
Between 1971 and 1991, deaths attributed to flames and hot liquids dropped an estimated 40% while deaths from smoke inhalation decreased only 12%.
71. OUTCOME Causes of improvement
Early and effective resuscitation
Control of sepsis
Improved management of inhalational injury, ventilation, ICU care.
Early excision
Development of alternative wound closure material
Nutrition
Team approach
72. INFECTION Infection is the leading cause of death in burn patients.
Risk factors :
1.Burns > 30% TBSA
2.Deep partial / full thickness burns
3.Age: under 16 or over 60 years
4.Preexisting disease; DM, malnutrition,..
73. INFECTION
During the past 15 years the primary cause of death has shifted from invasive burn wound infections to bronchopneumonia.
74. Wound Care Cleaning
Debriding
Topical Antimicrobial
Protection
Evaluation
75. Surgical Therapy Sharp Debridement (tangential, epifascial)
Split Thickness Skin Grafts
Alternative Wound Coverage
76. PRINCIPLES OF BURN RECONSTRUCTION
Direct closure
Adjacent tissue transfer
Skin grafts; FTSG vs STSG
Cultured epithelial autograts
Flaps
Tissue expansion
79. FULL THICKNESS BURN
80. 21 DAYS LATER
81. 21 DAYS LATER
82. ONE YEAR LATER