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Heat Emergencies. Chapter 193. Physiologic Response to Heat. Dilatation of the blood vessels (esp skin) Increased sweat production Decreased heat production Behavioral heat control Long-term: Acclimatization. Path to Heat Injury. Increased heat production
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Heat Emergencies Chapter 193
Physiologic Response to Heat Dilatation of the blood vessels (esp skin) Increased sweat production Decreased heat production Behavioral heat control Long-term: Acclimatization
Path to Heat Injury • Increased heat production • Obesity, skin conditions, febrile illness, medications • Increased external heat gain • Confinement hyperpyrexia • Decreased heat loss • Exertional heat injury, overdressed
Clinical Features Heat Edema Prickly Heat Heat Cramps Heat Tetany Heat Syncope Heat Exhaustion Heat Stroke
Heat Stroke • Core temp >40*C, CNS dysfunction & anhidrosis • Treatment: • ABC, BGL, O2, Cardiac monitoring, LR @ 250mL/h • Cooling Techniques: • Evaporative; ice packs to axillae & groin; immersion • Stop when core temp reaches 40*C (104F) • Complications: • Pulmonary edema, electrolyte abnl, thermal injury to liver, hematologic disorders, & renal damage
Disposition Minor heat illnesses may be treated in ED and discharged Heat stroke requires ICU admission Consider comorbidities when dispositioning heat exhaustion