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BLUNT OCULAR TRAUMA. Dr Soujanya K. general rule. anterior segment, or the posterior half is preferentially affected. Delayed or progressive changes: in some cases Guarded visual prognosis for all cases Review for months to years. Mechanics of concussion injuries. Coup or Direct damage.
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BLUNT OCULAR TRAUMA Dr Soujanya K
general rule anterior segment, or the posterior half is preferentially affected.
Delayed or progressive changes: in some cases • Guarded visual prognosis for all cases • Review for months to years
Coup or Direct damage max. damage at the site of impact
Contre-coup damage max. damage at a point distant from the actual site of impact
Blunt injury Mechanism of injury Dr UmaKulkarni
Blunt injury Mechanism of injury Dr UmaKulkarni
Blunt injury Inside-out injury Mechanism of injury Dr UmaKulkarni
A, direct impact; B, compression wave force; C, reflected compression wave; D, rebound compression wave.
subconjunctivalhaemorrhage: no treatment.
CORNEA Abration > distortion of corneal reflex > flouresceinstain > acute pain & lacrimation
RECURRENT EROSIONS -Spontaneous or injury with babies’ fingernails. -Heals and recurs after days,weeks or months. - occurs on waking up in the morning - Epithelium loosely attached to the Bowmans membrane
Treatment >Debridethe loose epithelium and > pad the eye for 48hrs or bandage soft contact lens.
Blood Staining Of Cornea >colourdep [reddish brown or greenish ]-On duration >clears slowly from periphery
CORNEAL OPACITY • Stromal edema • DM folds Stromal edema, DM Folds DM Tear
Sclera • Partial thickness scleral wounds (lamellar scleral lacerations)
Iris Traumatic miosis
TRAUMATIC MYDRIASIS Large and immobile pupil minute ruptures in the pupillary margin
IRIDODIALYSIS • Black biconvex area at the periphery • D shaped pupil • Distant direct O’scopy- red glow in the periphery • Zonules may also be seen • Monocular Diplopia is a common complaint
ANTEFLEXION OF THE IRIS Pigmented back of the iris faces forwards
Retroflexion of the iris • Whole of the iris is doubled back into the ciliary region and becomes invisible.
ANGLE RECESSION GLAUCOMA • Traumatic secondary open angle glaucoma • The ciliary body is torn- • longitudinal muscle remains attached to the spur at its insertion • circular muscle, pars plicata and the iris root are displaced posteriorly
Clinical features • Unilaterally raised IOP • Abnormally deep AC in the involved meridian • Hyhaema • Gonioscopy- Irregularly broad CB bandassociated with changes of optic neuropathy.
Circular ring of brown pigment –on ant. capsule. • Due to striking of the contracted pupillary margin against the crystalline lens. • It is always smaller than the size of the pupil.
Concussion cataract • Imbibition of aqueous • Direct mechanical
Early Rosette Cataract Feathery lines of opacities along the star-shaped suture lines; usually in the posterior cortex
LATE ROSETTE CATARACT • Posterior cortex, 1 to 2 years after the injury. • Its sutural extensions are shorter and more compact
Discrete subepithelial opacities • Traumatic zonular cataract. • Early maturation of senile cataract • Traumatic absorption of the lens