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Arrhythmias. Rhythms and Arrhythmia. SA Node Normal sinus rhythm Sinus Bradycardia Sinus Tachycardia Sick Sinus Syndrome Atria Ectopic beats Fibrillation Flutter Supraventricular tachycardia. Rhythms and Arrhythmia. AV Node Reentrant tachycardia 1 st 2 nd 3 rd degree blocks
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Rhythms and Arrhythmia • SA Node • Normal sinus rhythm • Sinus Bradycardia • Sinus Tachycardia • Sick Sinus Syndrome • Atria • Ectopic beats • Fibrillation • Flutter • Supraventricular tachycardia
Rhythms and Arrhythmia • AV Node • Reentrant tachycardia • 1st 2nd 3rd degree blocks • Bundle branch block • Ventricles • Ectopic beats • Tachycardia • Preexitation • Ischemia
Normal Sinus Rhythm • Normal sequence of conduction, originating in the sinus node and proceeding to the ventricles via the AV node and His-Purkinje system. • EKG Characteristics: Regular narrow-complex rhythm
Sinus Tachycardia Aetiologies • Fever • Hyperthyroidism • Hypovolaemia • Anxiety • Pheochromocytoma • Sepsis • Anaemia • Exposure to stimulants (nicotine, caffeine) or illicit drugs (amphetamines) • Hypotension and shock • Pulmonary embolism • Acute coronary ischemia and myocardial infarction • Heart failure • Chronic pulmonary disease • Hypoxia http://www.youtube.com/watch?v=zbDtMtJyVtI
Sinus Bradycardia • Sinus Bradycardia • HR< 60 bpm • QRS is narrow and preceded by p wave • Can be normal in well-conditioned athletes at rest or children during sleep • So when is it not normal? • Is not normal if the sinus rhythm can’t increase with exercise
Sinus Bradycardia - aetiologies • Normal aging • 15-25% following AMI • Sick sinus syndrome • Hypothyroidism • Hypothermia • Hypokalemia • Situational: micturation, coughing • Drugs: beta-blockers, digoxin, calcium channel blockers, amiodarone, lithium http://www.youtube.com/watch?v=Yff9VvNGL5w&feature=related
Sick Sinus Syndrome • Aetiology • Fibrosis • Atheroschlerosis of RC artery • SLE, collagen vasc diseases • Chagas disease - yeay • Injury, heart surgery • Infiltrative diseases • sarcoid • amyloid • Symptoms • Weakness • Palpitations • Syncope
Disorders of the SA Node • Sinus arrest • Tachycardia-bradycardia syndrome • Alternating brady-tachy causing palpitations • SA exit block • Normal SA impulse but conduction to atria is impaired
Atrial Ectopic Beats • Ectopic P waves look abnormal • SA node continues regardless in compensatory delay • SA node timing is reset if premature SA firing caused by ectopic – non compensatory
Atrial Fibrillation • Constant conduction within the atria, multiple circuits, loss of contractility • Can be paroxysmal, persistent or permanent • 1 in 5 strokes caused by AF: patients require anticoagulation therapy to prevent clot formation
Atrial Flutter • Aetiology • 30% have no underlying cardiovascular disease • 30% CAD • 30% hypertension • Pathophysilogy • Single reentrant circuit around the tricuspid valve • Distinctive sawtooth ECG
SupraventricularTachycardias • Any tachycardic rhythm originating above the ventricles • It usually involves ectopic pacemaker cells or an accessory (reentrant) pathway • http://www.youtube.com/watch?v=Y7QdOBYeAS4
Mechanism of Reentry • A: slow conduction, short RP • B: normal conduction and RP
AV Node Reentrant Tachycardia • Aetiology • Formation of “beta” pathway withinor alongside the node and exacerbated by • Anxiety • Exertion • Caffeine, alcohol, drugs • Signs & Symptoms • Palpitation • Dizziness, syncope • Chest pain, angina (if coexisting CAD)
First Degree Block • If the PR interval is more than 1 large square (0.2s) then this is a 1st degree AV block • Not associated with morbidity or mortality but foreshadows a more severe block if MI occurs • Risk factor = age
Second Degree Block • A conduction delay within the AV node causing increasingly lengthened PR intervals until the node cannot transmit the signal to the ventricles (the Wenkebachphenominum) • Aetiology • Structural heart disease • Drugs: digoxin, Na, beta and Ca channel blockers, tricyclic antidepressants & lithium at toxic levels • Metabolic: hyperkalaemia, hypermagnesaemia, hyperthyroidism, Addison’s • Enhanced vagal tone due to pain, athletes at rest • A shit load more at http://emedicine.medscape.com/article/161919-overview • Mostly intranodal but poor prognosis if infranodal
Second Degree Block Type I Wenckebach: P waves shown by arrows, lengthening PR interval Type II: P waves shown by arrows, unpredictable non-conduction and loss of QRS
Third Degree Heart Block • Complete interruption of conduction from the atria • Ventricles show escape rhythms • Aetiology • Infectious: Endocarditis, rheumatic fever • Neuromuscular: muscular dystrophy • Drugs (see 2nd degree HB) • Rheumatic, infiltrative, metabolic, electrolyte ...
BBB • Normal P waves • Normal PR interval • Widening of QRS to more than 0.12s because of delayed depolarisation of the ventricle
Ventricular Ectopic BEats • Premature beats originating within the myocardium • Distinctive tall, wide beats precede the P wave
VT • Reentrant arrhythmia as a result of scarring of the myocardium • The rhythm can stabilise, revert spontaneously to sinus rhythm or lead to VF and death
Ventricular Preexitation • Associated with WPW syndrome • Accessory pathway Bundle of Kent allows early ventricular depolarisation forming a blunted Q wave called a delta wave and an abnormal T wave (last 2 beats below)
Myocardial Ischemia • Myocardial ischemia caused by narrowing of the coronary arteries results in ST depression • Below the depression is shown in the shaded areas
ECG Online Test • http://www.andrews.edu/~schriste/HealthTeaching/Practice/Sinus_and_Atrial/si01.html