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AR in VSD - mechanisms

AR in VSD - mechanisms. DESSERT SESSION Dr SHREETAL RAJAN NAIR. AR in VSD. 1. Perimembranous defects : 5 – 8% ; in whites 2. Doubly committed VSD : - 30% ; in A sian descent - Male predominance (2:1). AR in VSD. AR rarely begins before 2 years and after 10 years

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AR in VSD - mechanisms

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  1. AR in VSD - mechanisms DESSERT SESSION Dr SHREETAL RAJAN NAIR

  2. AR in VSD • 1. Perimembranous defects : • 5 – 8% ; in whites 2. Doubly committed VSD : - 30% ; in Asian descent - Male predominance (2:1)

  3. AR in VSD • AR rarely begins before 2 years and after 10 years • Peak incidence noted between 5-9 years

  4. Pathogenesis • Major features : a. lack of anatomic support for valve - deficiency of conal septum - defect in sinus of valsalva - defect in aortic valve commissures b. hemodynamic effects c. Bicuspid aortic valve (mechanism unknown) Pathogenetic Mechanisms of Prolapsing Aortic Valve and Aortic Regurgitation Associated With Ventricular Septal Defect : Anatomical, Angiographic, and Surgical Considerations By KATSUHIKo TATSUNO, M.D., Souji KONNO, M.D., MASAHIKO ANDO, M.D., AND SHIGERU SAKAKIBARA, M.D.

  5. Pathogenetic Mechanisms of Prolapsing Aortic Valve and Aortic Regurgitation Associated With Ventricular Septal Defect : Anatomical, Angiographic, and Surgical Considerations By KATSUHIKo TATSUNO, M.D., Souji KONNO, M.D., MASAHIKO ANDO, M.D., AND SHIGERU SAKAKIBARA, M.D.

  6. Thank you

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