Figure 1 Putative evolution of OA phenotypes and therapies

Sep 17, 2014

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Figure 1 Putative evolution of OA phenotypes and therapies. Conaghan, P. G . (2012) Parallel evolution of OA phenotypes and therapies Nat. Rev. Rheumatol. doi:10.1038/nrrheum.2012.225.

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Figure 1 Putative evolution of OA phenotypes and therapies

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Figure 1Putative evolution of OA phenotypes and therapies Conaghan, P. G.(2012)Parallel evolution of OA phenotypes and therapies Nat. Rev. Rheumatol. doi:10.1038/nrrheum.2012.225

Pros and Cons of Historic Therapies for OA and RA

Comprehensive Management of OA. Creamer et al. Lancet. 1997;350:503-508.Gilbert. Am J Knee Surg. 1998;11:42-46.Hochberg et al. Arthritis Rheum. 1995;38:1535-1540.. . . . . . . NonpharmacologicPatient educationPsychosocial support Physical/occupational therapyWeight reduction Exercise . . . PharmacologicAnalgesicsNSAIDsTopical creamsGlucocorticoidsHyaluronic acidExperimental therapies.

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Chapter 7 Beyond alleles: quantitative genetics and the evolution of phenotypes

Chapter 7 Beyond alleles: quantitative genetics and the evolution of phenotypes. Continuous traits have a complex genetic basis. Polygenic trait: influenced by many genetic loci Interaction between alleles (epistasis) Interaction with environment (phenotypic plasticity)

651 views • 37 slides

1. From Genes to Phenotypes

1. From Genes to Phenotypes. Mendel was fortunate to have chosen some of the most genetically simple of characters in the garden pea for his seminal experiments that laid the foundation for the science of genetics.

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DOK 1 5.OA.1

I can evaluate numerical expressions using the order of operations including parentheses, brackets, or braces. DOK 1 5.OA.1. I can write numerical expressions for given numbers with operations words. DOK 1,2 5.OA.2. I can interpret numerical expressions without solving. DOK 1,2 5.OA.2.

862 views • 63 slides

Evolution: Natural Selection and Phenotypes

Evolution: Natural Selection and Phenotypes. Big Idea 1: Process of evolution drives the diversity of life. Essential Knowledge. 1A1: Natural selection is a major mechanism of evolution. 1A2: Natural selection acts on phenotypic variations.

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5.OA.1

Use parentheses, brackets, or braces in numerical expressions, and evaluate expressions with these symbols. 5.OA.1.

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4.OA.1

Interpret a multiplication equation as a comparison, e.g., interpret 35 = 5 x 7 as a statement that 35 is 5 times as many as 7 and 7 times as many as 5. Represent verbal statements of multiplicative comparisons as multiplication equations. 4.OA.1.

1.04k views • 39 slides

Figure 1

LFA classes, go here. Figure 1. Find the month and class, then based on class size, see if there are openings. If the class is full, you can register (and place) the person on a wait list. This is a good option, you just have to check it regularly.

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Topic 16. Lecture 25. Origin of Life and Evolution of Functional Phenotypes

Topic 16. Lecture 25. Origin of Life and Evolution of Functional Phenotypes We have no direct data, and only rather speculative ideas, on how life emerged. The context of the origin of life:

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Figure 1

a. d. b. e. c. f. Effect of different induction strength of one chaperone team at constant induction strength of the other chaperone. Figure 1. 80 66 47 40 35 29 17. 1 2 3 4. STD Streptokinase. Degraded Fragments ~35 kDa.

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Figure 4 The putative FLCN pathway

Figure 4 The putative FLCN pathway. Linehan, W. M. et al. (2010) The genetic basis of kidney cancer: a metabolic disease Nat. Rev. Urol. doi:10.1038/nrurol.2010.47.

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SNOMED and “phenotypes”

SNOMED and “phenotypes”. Signs, symptoms, findings, etc. Signs, Symptoms and Findings: Steps Toward an Ontology of Clinical Phenotypes Sept 3-4, 2008 Dallas TX. Symptoms & signs. Conclusions of the SNOMED RT group (1996):

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CPC WinCC OA evolution

CPC WinCC OA evolution. Acknowledgments. Herve Milcent , EN-ICE-SIC UNICOS Cyril Caillaba , EN-ICE-SCD unPackageCreation Bartlomiej Urbaniec , EN-ICE-SIC Flex Extractor Axel Voitier , EN-ICE-SCD PVSSBootstrapper Marco Boccioli , EN-ICE-SCD testing/review. Presentation plan.

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Figure 1.

Figure 1. a. c. b. d. Figure 2. a. e. b. f. c. g. d. h. Figure 3. B. Figure 4. Figure 5.

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Figure 1

LiuPan mountains. Figure 1. Figure 2. 20. 15. 10. Species. S obs. 5. Michaelis-Menten. Chao1. Jackknife2. 0. 0. 20. 40. 60. 80. 100. Samples. Figure 3. Afforestation gradient. Deforestation gradient. Afforested set-aside fields. Woody shrub. Non-woody shrub.

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Putative pheoviral replication

HGT. H. 2 OBPs. Polydnavirus. replication. Organism. E. Mimivirus. 2 OBPs. A. Ancestral NCLDV. 2 OBPs. 1 OBP. F. Giant DNA virus. Herpes virus lineage. D. Herpes virus, Asfarvirus. HGT. Integration of one copy of the viral genome. C. 2 OBPs. Primitive eukaryotic cell.

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1.OA.1

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Figure 1

Figure 1.

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Figure 1 Evolution of clinical genetic screening for hereditary cancers

Figure 1 Evolution of clinical genetic screening for hereditary cancers. Samuel, N. et al. ( 2014) Management of familial cancer: sequencing, surveillance and society Nat. Rev. Clin. Oncol. doi:10.1038/nrclinonc.2014.169.

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Table 1 Connectivity correlates of clinical phenotypes

Table 1 Connectivity correlates of clinical phenotypes. Pievani, M. et al. (2014) Brain connectivity in neurodegenerative diseases—from phenotype to proteinopathy Nat. Rev. Neurol. doi:10.1038/nrneurol.2014.178.

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Content Based Assessment for 5.OA.1 and OA.2

Name____________________ Date ___________________. Content Based Assessment for 5.OA.1 and OA.2. Find the missing number. Tell how you arrived at the answer. 1. 4 x 6 = x 3. Find the missing number. Tell how you arrived at the answer. 2. 12 x 15 = x 30.

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