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Practical Electrocardiography - Rate and Rhythm -. Scott E. Ewing, D.O. Cardiology Fellow Lecture #2. Review. Electrophysiology Anatomy Depolarization ECG Paper Lead Placement Normal ECG Waves / Intervals / Segments. Depolarization. ECG Frontal Plane. Normal ECG. QRS Complex.
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Practical Electrocardiography- Rate and Rhythm - Scott E. Ewing, D.O. Cardiology Fellow Lecture #2
Review • Electrophysiology • Anatomy • Depolarization • ECG Paper • Lead Placement • Normal ECG • Waves / Intervals / Segments
8-Step Method ECG Interpretation • Rate • Rhythm • Axis • P wave • PR interval • QRS complex • QT interval • ST segment and T wave
Rhythm • Atrial • Junctional • Ventricular • Pacemaker • Last but not least
Sinus Bradycardia • Pathophysiology • Increased vagal tone in athletes • Inferior wall myocardial infarction • Digitalis glycosides, beta-blockers, calcium channel-blocking agents, class I antiarrhythmic agents, amiodarone • Other drugs, toxins, environmental exposure (lithium, paclitaxel, toluene, dimethyl sulfoxide, topical ophthalmic acetylcholine, fentanyl, reserpine, clonidine) • Electrolyte disorders • Infection (diphtheria, rheumatic fever, viral myocarditis) • Sleep apnea • Hypoglycemia • Hypothyroidism • Hypothermia • Increased intracranial pressure
Sinus Tachycardia • Pathophysiology • Hypoxia • Hypovolemia / Sepsis • Pain • Fever • Anxiety • Hyperthyroidism • PE • Exercise • Drugs (nicotine, caffeine, atropine, pseudoephedrine, cocaine, methamphetamines, ecstasy)
1st Degree AV Block • Pathophysiology • PR interval represents time needed for electrical impulse from sinoatrial node to conduct through the atria, AV node, bundle of His, bundle branches, and Purkinje fibers • PR interval prolongation due to conduction delay within the right atrium, the AV node, or the His-Purkinje system • AV nodal dysfunction accounts for the majority of cases • First-degree AV block caused by conduction delay in the His-Purkinje system often is associated with bundle-branch block
Karel Frederik Wenckebach(1864 – 1940) • 1988 – Doctorate University of Utrecht, Netherlands • 1901-1910 – professor of IM at Groningen, Netherlands • 1911-1914 – professor of IM Strasbourg , France • 1915-1929 – professor of IM Vienna, Austria, retired from his chair 1929 • Early work concerned embryology, later pathology and clinics of heart and circulatory diseases • 1903-1904 – first description of the beneficial effects of quinine alkaloids on arrhythmias and mainly in patients with auricular fibrillation or recent onset • 1905-1906 – second degree AV block independently discovered by English physician John Hay and Wenckebach • 1934 – monograph on beriberi
2nd Degree AV BlockMobitz Type I (Wenckebach) • Pathophysiology • Conduction disturbance in the AV node • Rarely secondary to AV nodal structural abnormalities when the QRS complex is narrow in width and no underlying cardiac disease is present • May be vagally mediated (well-trained athletes, digoxin excess, neurally mediated syncopal syndromes) • Vagally mediated AV block improves with exercise and may occur more commonly during sleep when parasympathetic tone dominates • Cardioactive drugs (digoxin, beta-blockers, calcium channel blockers, and certain antiarrhythmic drugs) • Various inflammatory, infiltrative, metabolic, endocrine, and collagen vascular disorders
2nd Degree AV BlockMobitz Type II (Hay) • Intermittent failure of conduction of P waves • PR interval is constant (may be normal or prolonged) • May include wide QRS • May progress to complete third degree AV block
Woldemar Mobitz(1889 – 1951) • Born May 31, 1889 St. Petersburg, Russia, the son of a prominent surgeon • 1908 – gymnasium Meiningen, Saxony • 1914 – doctorate University of Munich • Internship, hospital service, and assistant years in the surgical clinics in Berlin and Halle, and medical clinics Munich and Freiburg • 1924 – first classified second degree AV block into Type I and II • 1928-1943 – professor extraordinary at University of Freiburg in Breisgau • Remained in Magdeburg until it was occupied by the Russian army in 1945 • Suffered from laryngeal tuberculosis until his death April 11, 1951 • Primary interest in cardiovascular circulation and arrhythmias
3rd Degree Heart Block • Pathophysiology • Class Ia antiarrhythmics (eg, quinidine, procainamide, disopyramide) • Class Ic antiarrhythmics (eg, flecainide, encainide, propafenone) • Class II antiarrhythmics (beta-blockers) • Class III antiarrhythmics (eg, amiodarone, sotalol, dofetilide, ibutilide) • Class IV antiarrhythmics (calcium channel blockers) • Digoxin or other cardiac glycosides • Infection • Profound hypervagotonicity • Anterior wall MI • Cardiomyopathy, eg, Lyme carditis and acute rheumatic fever • Metabolic disturbances, eg, severe hyperkalemia