1 / 28

Cerebral Palsy

Cerebral Palsy. Definition: It is static non progressive disorder of posture and movement resulting from defect or lesion of the developing brain 2/1,000 population First described almost 150 yrs ago by Little ,an orthopedic surgeon. Association of CP. Association of CP-

gabi
Download Presentation

Cerebral Palsy

An Image/Link below is provided (as is) to download presentation Download Policy: Content on the Website is provided to you AS IS for your information and personal use and may not be sold / licensed / shared on other websites without getting consent from its author. Content is provided to you AS IS for your information and personal use only. Download presentation by click this link. While downloading, if for some reason you are not able to download a presentation, the publisher may have deleted the file from their server. During download, if you can't get a presentation, the file might be deleted by the publisher.

E N D

Presentation Transcript


  1. Cerebral Palsy • Definition: It is static non progressive disorder of posture and movement resulting from defect or lesion of the developing brain • 2/1,000 population • First described almost 150 yrs ago by Little ,an orthopedic surgeon

  2. Association of CP • Association of CP- ■MR ■ Epilepsy-25-35% ■ Speech defect 50% ■ Deafness-10% ■ Visual defect-20% ■ Dental problem ■ Behavioral disorder 40% ■ Orthopedic problem ■ Drooling

  3. Classification • Physiologic: • Spastic (65%) ■ Athetoid (30%) • Rigidity ■ Ataxia • Tremor ■ Atonic/hypotonic • Mixed ■ unclassified

  4. Anatomical classification • Monoplegia ■ Paraplegia • Hemiplegia ■ Triplegia • Quadriplegia ■ Diplegia • Double hemiplegia

  5. Etiological classification • Prenatal: ■ Congenital anomalies ■ Maternal: – Torch infection,Chorioamnionitis, Maternal sepsis, UTI Temperature during labor Toxemias of pregnancy

  6. Perinatal Causes • Birth trauma • Perinatal asphyxia <10% • LBW babies (prematurity IUGR)-due to intracerebral hge, periventricular leukomalacia

  7. Postnatal causes • Infections-Meningitis, Encephalitis • Trauma- Head injury, Subdural hemotoma • Toxic – kernicterus • Cerebrovascular -thrombosis of C.vessels • Endocrine and metabolic- hypothyroidism, hypoglycemia, hypocalcemia, dyselectrolyte • Gross PEM in early infancy

  8. Functional classification • Class I- No limitation of activity • Class II- Slight to moderate limitation (20%) • Class III- Moderate to great limitation(50%) • Class IV – No useful physical activity (30%)

  9. Clinical manifestations • Common presentation : • Delayed milestones with indifferent look • Fisting of hands with extended extremities • Microcephaly, Mental retardation • Behavioral abnormalities • Visual, hearing, speech defects

  10. Common Presentations • persistence of NN reflexes , • Exaggerated jerks • Adductor spasms • gait –tip toe • Epilepsy

  11. Spastic hemiplegia • Arms often more involved than leg-difficulty in hand manipulation is obvious by 1 yr • Delayed walking -18-24 mo • Equinovarus deformity of foot, walks on tip toes because of increased tone • Affected upper limbs has dystonic posture when child runs

  12. Spastic hemiplegia • Deep tendon reflexes increased, ankle clonus, babinski sign + • 1/3rd have seizure disorder • 25% have MR • CT/MRI- atrophic cerebral hemisphere with dilated lateral ventriclecontralateral to the affected side

  13. Spastic Diplegia • Bilateral spasticity of legs • 1st noticed when infant begins to crawl-tends to drag the legs behind more ( commando crawl) • Severe spasticity –application of diaper is difficult due to excess adduction of hips • Brisk reflexes, ankle clonus • Scissoring posture of lower extremity when suspended by axilla

  14. Spastic Diplegia • Walking tiptoes, disuse atropy ,impaired growth of lower extremity • Intellectual development normal • Minimal seizures • CT/MRI-periventricular leukomalacia of white matter mainly lower limb fibres

  15. Spastic quadriplegia • Most severe form ,most common • All extremities severely impaired • High association with MR and seizure • Supranuclear bulbar palsies+--aspiration pneumonia • Flexion Contractures of knees and elbows • Spastic quadriplegia + athetosis +mixed CP

  16. Athetoid CP • Relatively rare these days due to aggressive management of hyperbilirubinemia • H/o NNJ += hypermyelination of basal ganglia called status marmoratus • Initially hypotonic, poor head control,marked head lag,feeding difficulty,drooling +

  17. Athetoid CP • After age 1yr –athetoid movements become evident • Speech is affected (slurred, voice modulation impaired) due to involvement of oropharyngeal muscles • Upper motor neuron signs –not present • Seizure uncommon • Intellect -preserved

  18. Rigidity/Ataxia/Tremor • Uncommon variety • Features og extrapyramidal lesion • Lead pipe or cogwheel type • Always associated with MR • Ataxia: • Mostly congenital due to cerebellar malformation • Tremor- constant severe coarse tremor

  19. Diagnosis • History • Examination • Serology-TORCH/VDRL • X-ray skull-intracranial calcification • EEG • Ct/MRI • Test of hearing ,vision • IQ test

  20. Treatment • Multidisciplinary approach –pediatrician playing the main role • Physiotherapist Orthopedic surgeon • Speech therapist ENT surgeon • Neurologist Social worker • Developmental psychologist

  21. Treatment • Counselling-teach parents-daily activities like feeding, carrying, dressing, bathing, playing, physiotherapy to limit abnormal muscle tone • Physiotherapy :- For arms-physio to start by age 6 months For legs-for effective weight bearing and wt transfer

  22. Treatment • Grasping ,release movements of hands, reciprocal movements of feet for walking, vocalised breathing for speech, parallel walking bars bicycles, special chair, grasping and releasing games • Massage, exercise and hydrotherapy

  23. Treatment • Adaptive equipments-walkers, poles standing frames, motorized wheel chair, special feeding devices, modified typewriters • Communication skills- use of symbols, specially adapted computers

  24. Treatment • Ophthalmologist-strbismus, nystagmus, optic atrophy • Orthopedics :- Hip, knee contractures-surgical release • Occupational therapy:- Simple movements for self help- feeding, dressing

  25. Treatment • Educational therapy: • Mild MR – ordinary school • Severe MR, severe disabilities-special school • Social therapy- social and emotional support to family • Rehabilitation and vocational guidance

  26. Treatment • Symptomatic- anticonvulsants for seizures • Muscle relaxants- benzodiazapines ,dantrolene sodium, Baclofen • Preventable causes shoud be prevented

  27. Prognosis • 10-50%-have seizure • Squint-50%,visual handicap-30% optic atrophy and cortical blindness • Walk by age of three, if not then unlikely that useful function will be gained

More Related