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Inflammation p.1. SYLLABUS: Robbins and Cotran , Pathologic Basis of Disease , Chapter : Acute and Chronic Inflammation (al l). Inflammation p.1. 103 Lobar pneumonia 106 Appendicitis 15 5 Nasal polyp Pericarditis (fribrionous) Follicular hyperplasia of the lymph node
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Inflammation p.1 SYLLABUS: Robbinsand Cotran, PathologicBasis of Disease, Chapter: Acuteand ChronicInflammation(all)
Inflammation p.1 103Lobar pneumonia 106Appendicitis 155 Nasal polyp • Pericarditis (fribrionous) • Follicular hyperplasia of the lymph node 119Candidiasis (esophagus)
Lobar pneumonia • alveoli filled with fibrinous exsudate • (fibrin stain)
Appendicitis • neutrophilic inflammatory infiltration of all the layers of appendix • possible mucosal ulcerations, inflammatory exsudate in the lumen of appendix, necrosis of the wall extending to serosa
Nasal polyp • edematous mucosa with a loose stroma • often hyperplastic or cystic mucous glands • a variety of inflammatory cells, including neutrophils, eosinophils, and plasma cells with occasional clusters of lymphocytes
Pericarditis (fibrinous inflammation) • fibrinous exsudate (eosinophilic meshwork of threads and amorphous masses) covering pericardium
Follicular hyperplasia of the lymph node (Reactive lymphoid hyperplasia, follicular pattern) • increase in number and size of germinal centers, due to expansion of follicular center, occasionally of mantle as well • follicular polarization retained
Candidiasis (esophagus) Organisms: a mixture of budding yeast forms (blastoconidia) and pseudohyphae, tubular structures separated by narrowed areas Tissue reaction: usually purulent but may be minimal or granulomatous