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ORAL CAVITY. DEVELOPMENTAL ANOMALIES 1. FACIAL CLEFTS 2. MACROGLOSSIA 3. MICROGLOSSIA 4. AGLOSSIA. CLEFT LIP. MACROGLOSIA. ORAL CAVITY. 5. FISSURED TONGUE 6. BIFID TONGUE 7.TONGUE TIE- SHORT LINGUAL FRAENUM 8.HAIRY TONGUE- HYPERTROPHIED
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ORAL CAVITY DEVELOPMENTAL ANOMALIES 1. FACIAL CLEFTS 2. MACROGLOSSIA 3. MICROGLOSSIA 4. AGLOSSIA
ORAL CAVITY 5. FISSURED TONGUE 6. BIFID TONGUE 7.TONGUE TIE- SHORT LINGUAL FRAENUM 8.HAIRY TONGUE- HYPERTROPHIED FILIFORM PAPILLAE - STAINED BLACK,BROWN,YELLOW WHITE BY FOOD,TOBACCO etc
INFLAMMATORY DISEASES OF ORAL CAVITY - STOMATITIS INFLAMMATION OF ORAL MUCOSA. 1. APHTHOUS ULCERS(CANKER SORES) ETIOLOGY UNKNOWN PRECIPITATED BY EMOTIONAL FACTORS,STRESS,ALLERGY ,HORMONAL IMBALANCE,TRAUMA ,NUTRITIONAL DEFICIENCIES, GIT DISTURBANCES etc
1. APHTHOUS ULCERS PAINFUL ORAL ULCERS 1cm OR MORE IN SIZE,HEAL WITHOUT SCAR FORMATION
INFLAMMATORY DISEASES OF ORAL CAVITY - STOMATITIS 2. HERPETIC STOMATITIS.
INFLAMMATORY DISEASES OF ORAL CAVITY - STOMATITIS 3. NECROTISING STOMATITIS(NOMA OR CANCRUMORIS---) POORLY NOURISHED CHILDREN, KWASHIORKER,INFECTIOUS DISEASES(MEASELES),IMMUNODEFICI ENCIES,
3.NECROTISING STOMATITIS(NOMA OR CANCRUMORIS---) ETIOLOGY EMOTIONAL STRESS CHARACTERISED BY NECROSIS OF THE MARGINAL GINGIVA
3.NECROTISING STOMATITIS(NOMA OR CANCRUMORIS---) EXTENDING TO ORAL MUCOSA CAUSING CELLULITIS OF THE TISSUE OF THE CHEEK. MAY PROGRESS TO GANGRENE OF THE CHEEK.
STOMATITIS 4. MYCOTIC INFECTIONS- ACTINOMYCOSIS , CANDIDIASIS
GLOSSITIS ACUTE GLOSSITIS- MEASELS,SCARLET FEVER- SWOLLEN PAPILLAE WITH ERUPTIONS.
STOMATITIS CHRONIC GLOSSITIS-RED RAW WITHOUT SWOLLEN PAPILLAE - MALNUTRITION ,SEEN IN PELLEGRA (NIACIN DEFICIENCY-vitaminB3 ) ARIBOFLAVINOSIS(vitamin B2)
STOMATITIS CHRONIC ATROPHIC GLOSSITIS- IN IRON DEFICIENCY,PERNICIOUS ANAEMIA ATROPHIED PAPILLAE & SMOOTH RAW TONGUE.
SYPHILITIC LESIONS PRIMARY - CHANCRE SECONDARY - MACULOPAPULAR ERUPTIONS TERTIARY - GUMMAS ORDIFFUSE FIBROSIS CONGENITAL SYPHILIS - FISSURES AT THE ANGLES OF MOUTH , PEG SHAPED NOTCHED HUTCHINSON’S INCISORS.
TUBERCULOUS LESIONS ULCERS OR ELEVATED NODULES
HIV INFECTION OPPORTUNISTIC INFECTIONS LIKE CANDIDIASIS HAIRY LEUKOPLAKIA - HAIRY OR CORRUGATED SURFACE), MALIGNANCY - KAPOSI’S SARCOMA
PIGMENTATY LESIONS SEEN IN SYSTEMIC & METABOLIC DISORDERS
PIGMENTATY LESIONS ADDISON’S DISEASE, ALBRIGHT SYNDROME (POLYOSTOTIC FIBROUS DYSPLASIA), PEUTZ –JEGHERS SYNDROME( CAFÉ –AU - LAIT SPOTS)
PIGMENTATY LESIONS HAEMOCHROMATOSIS, PIGMENTED NAEVI, MALIGNANT MELANOMA, EXOGENOUS PIGMENT LIKE LEAD SULFIDE.
ORAL SUBMUCOSAL FIBROSIS SEEN PARTICULARLY IN INDIA COLLAGEN THICKENING OF THE MUCOSA OF THE LIPS,CHEEKS & PALATE ACCOMPANIED BY LEUKOPLAKIA OF OVERLYING MUCOSA.
ORAL SUBMUCOSAL FIBROSIS CAUSES STIFFENING OF THE MOUTH PARTS. PERSON FIND IT DIFFICULT TO CHEW,DRINK, EAT ,TALK.
TUMOURS OF THE ORAL CAVITY BENIGN TUMOURS 1. SQUAMOUS PAPILLOMA 2.HAEMANGIOMA 3.LYMPHANGIOMA
TUMOURS OF THE ORAL CAVITY 4.FIBROMA 5.TUMOURS OF MINOR SALIVARY GLANDS.
TUMOUR LIKE LESIONS ORAL CAVITY EPULIS – 1.FIBROUS EPULIS –LESION OCCURING ON GINGIVA-IS A LOCALISED HYPERPLASIA OF CONNECTIVE TISSUE FOLLOWING TRAUMA OR INFLAMMATION
EPULIS ( BENIGN, ON GINGIVAL OR ALVEOLAR MUCOSA) 2.GIANT CELL EPULIS- A VARIANT, COMMONLY IN FEMALES-REACTIVE CHANGE TO TRAUMA WITH NUMEROUS OSTEOCLAST LIKE GIANT CELLS
EPULIS ( BENIGN, ON GINGIVAL OR ALVEOLAR MUCOSA) 3. CONGENITAL EPULIS –A BENIGN TUMOUR KNOWN AS GRANULAR CELL TUMOUR ARISING FROM NEURAL TISSUE
ORAL LEUKOPLAKIA(WHITE LESIONS) DEFINITION- WHITE PATCH OR PLAQUE ON THE ORAL MUCOSA EXCEEDING 5mm IN DIAMETER,WHICHCAN NOT BE RUBBED OFF NOR CANBE CLASSIFIED INTO ANY OTHERDIAGNOSABLE DISEASE.
ORAL LEUKOPLAKIA(WHITE LESIONS) EPITHELIAL THICKENING WHICH MAY RANGE FROM COMPLETELY BENIGN TO ATYPICAL TO PREMALIGNANT CELLULAR CHANGES.
LEUKOPLAKIA SITES OF PREDILECTION IN DESCENDING ORDER OF FREQUENCY- CHEEK, ANGLES OF MOUTH,ALVEOLAR MUCOSA,,TONGUE,LIP,HARD & SOFT PALATE,& FLOOR OF THE MOUTH
LEUKOPLAKIA NODULAR FORM IS MORE LIKELY TO PROGRESS TO MALIGNANCY.
ETIOLOGY OF LEUKOPLAKIA 1. TOBACCO IN VARIOUS FORMS CHEWING TOBACCO AS IN PAN ,PAN MASALA SMOKERS KERATOSIS( STOMATITIS NICOTINA)
ETIOLOGY OF LEUKOPLAKIA 2.CHRONIC FRICTION - ILLFITTINGDENTURES, JAGGED TEETH 3 .LOCAL IRRITANTS- SUCH AS ALCOHOL, SPICY HOT FOOD, BEVERAGES.
LEUKOPLAKIA 4.AIDS PATIENTS - HAIRY LEUKOPLAKIA- HAIRY OR CORRUGATED SURFACE PATHOLOGY 2 TYPES OF LEUKOPLAKIA 1.HYPERKERATOTIC- ORDERLY REGULAR HYPERPLASIA OF SQUAMOUS EPITHELIUM WITH HYPERKERATOSIS ON THE SURFACE.
PATHOLOGY OF LEUKOPLAKIA 2. DYSPLASTIC TYPE- ERREGULAR STRATIFICATION , ABNORMAL MITOSIS, HYPERCHROMATISM,PLEOMORPHISMLOSS OF POLARITY, INDIVIDUAL CELL KERATINIZATION.
LEUKOPLAKIA PROGNOSIS MILD DYSPLASIA REVERT BACK TO NORMAL WHEN OFFENDING AGENT IS REMOVED SEVERE DYSPLASIA- PROGRESS TO CARCINOMA.